In the United States in 1987 upon the death of his mother, electing not to remain with his father, who did not "prefer" him. Petitioner stayed with an aunt for three months, but, apparently not wanted there either, took to the street, where he became a drug user and heroin addict. Petitioner's life had no meaning. He wanted to die from an overdose of drugs. He turned to a life of crime in which Alex Torres was his mentor. Petitioner began visiting the Gama brothers, whom he referred to as "fags, " on a nearly every-day basis in order to obtain food and drugs, but apparently not sex. Petitioner went to the Gamas' apartment on February 3, 1991--he was not sure of the date--but no one answered. Petitioner continued his crime spree with Alex Torres through the remainder of that month. Petitioner contended that he did not kill the victims, that he did not understand "some English" when he signed the papers that were the sole reason for his current presence before the court. Petitioner stated that he was going to die in two or three years and requested that he be sent to the penitentiary for his remaining days. During cross-examination, Petitioner again maintained that he had not killed the victims. Petitioner was then asked whether he was sorry that he killed Jesus Gonzales. "Who's Jesus Gonzales?, " Petitioner asked. Petitioner was then reminded, and acknowledged, that Gonzales was his friend. After hearing argument, the trial court discussed its findings of fact and conclusions of law. The trial court initially cited the fact that Petitioner had killed two people and further, that the murders had occurred during the commission of a felony. The trial court also cited the Gonzales murder, which had been pursuant to a preconceived plan and for which Petitioner had agreed to receive money. The trial court stated that Petitioner was an intelligent person "not without cleverness in his answers, not with facility in his words" and stated that it believed that Petitioner understood 20.
But during the festival there is huge pressure on the local council to just get rid of the dogs from this area and this is done through the most unimaginable means. Once I found this out, I couldn't bear the thought that I would be there and know that this would be happening and not do something about it. So we are "doing something about it". We managed to get the Government to provide a suitable building and land where we can make pens that are completely dog proof. At first I thought that we would just catch all the dogs not an easy task in itself as they all hang around in their territorial groups, and mixing the, for instance, fusidic acid and betamethasone.
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18. Nilsberth C, Westlind-Danielsson A, Eckman CB, et al. The Arctic APP mutation E693G ; causes Alzheimer's disease by enhanced Abeta protofibril formation. Nat Neurosci. 2001; 4: 887-893. Snowden JS, Neary D, Mann DMA. Frontotemporal dementia. Br J Psychiatry. 2002; 180: 140-143. Galasko D, Marder K. Picking away at frontotemporal dementia. Neurology. 2002; 58: 1585-1586. Kaye JA. Diagnostic challenges in dementia. Neurology. 1998; 51 suppl 1 ; : 45-52. 22. The Lund and Manchester Groups. Clinical and neuropathological criteria for frontotemporal dementia. J Neurol Neurosurg Psychiatry. 1994; 57: 416-418. Neary D, Snowden JS, Gustafson L et al. Frontotemporal lobar degeneration: a consensus on clinical diagnostic criteria. Neurology. 1998; 51: 1546-1554. Goedert M. The significance of tau and -synuclein inclusions in neurodegenerative diseases. Curr Opin Genet Dev. 2001; 11: 343-351. Lee VMY, Goedert M, Trojanowski JQ. Neurodegenerative tauopathies. Annu Rev Neurosci. 2001; 24: 1121-1159. Grundke-Iqbal I, Iqbal K, Tung YC, Quinlan M, Wisniewski HM, Binder LI. Abnormal phosphorylation of the micortubule-associated protein tau tau ; in Alzheimer cytoskeletal pathology. Proc Natl Acad Sci U S A. 1986; 83: 4913-4917. McKeith IG. Dementia with Lewy bodies. Br J Psychiatry. 2002; 180: 144-147. Spillantini MG, Goedert M, Crowther RA, Murrell JR, Farlow MR, Ghetti B. Familial multiple system tauopathy with presenile dementia: a disease with abundant neuronal and glial tau filaments. Proc Natl Acad Sci U S A. 1997; 94: 4113-4118. Kosaka K. Dementia and neuropathology in Lewy body disease. Adv Neurol. 1993; 60: 456-463. Duda JE, Lee VM, Trojanowski JQ. Neuropathology of synuclein aggregates. J Neurosci Res. 2000; 15: 61121-61127. Weiner MF. Dementia associated with Lewy bodies: dilemmas and directions. Arch Neurol. 1999; 56: 1441-1442. Kahle PJ, Haass C, Kretschmar HA, Neumann M. Structure function of -synuclein in health and disease: rational development of animal models for Parkinson's and related diseases. J Neurochem. 2002; 82: 449-457, for example, betamethasone oral.
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Parallel trading means that drugs are bought from a wholesaler in one European country and then re-packed and distributed to pharmacies in other countries. In Sweden, Meda is one of the largest parallel trading companies through its subsidiary Cross Pharma AB. Sweden and Norway are prioritised in parallel trading. Parallel Trading achieved sales in 2001 amounting to SEK 482.2m 477.0 ; . Several new products have been introduced during the past year, and products which have become unprofitable due to the weakness of the Swedish krona have been phased out.
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Clinical reactions in allergic diseases such as rhinitis, urticaria, and food and drug allergies. The need for more effective and safe antihistamines is critical and intensive drug development has become more demanding and competitive. Although the old "first generation" antihistamines were effective, major limitations included their strong potential for sedation and their anti-cholinergic side effects. Not only could patients not function well in their normal daily activities, but these medications posed an important risk for safety, particularly for motor vehicle drivers and machine and precision instrument operators. Unacceptable side effects were a particular problem in the elderly. In May 2001, CONGA, an international consensus group, convened to formulate guidelines for the development of new antihistamines. Several important areas were reviewed and a document of recommendations was published, focusing specifically on the safety and efficacy aspects of antihistamines. 19. ANTITUSSIVES 905. Acute psychosis associated with Coricidin HBP Cough and Cold Medicine - Ginsberg D.L. [Dr. D.L. Ginsberg, Tisch Hospital, Department of Psychiatry, New York University Medical Center, New York City, NY, United States] - PRIM. PSYCHIATRY 2005 12 10 ; 20. DRUGS USED IN DERMATOLOGY 906. Management of guttate and generalized psoriasis vulgaris: Prospective randomized study - Caca-Biljanovska N.G. and V'Ickova-Laskoska M.T. [M.T. V'Ickova-Laskoska, University of Sts. Cyrilus and Methodius, School of Medicine, Department of Dermatology, Vodnjanska 17, 1000 Skopje, Macedonia] - CROAT. MED. J. 2002 43 6 ; - summ in ENGL Aim. To assess the efficacy of betamethasone dipropionate 0.05% cream plus ultraviolet B UVB ; radiation with and without additional penicillin therapy in the treatment of guttate psoriasis, and to compare the efficacies of oral psoralen plus ultraviolet A PUVA ; therapy and systemic retinoids therapy for treatment of generalized psoriasis. Methods. Sixty patients with guttate n 20 ; and generalized psoriasis vulgaris n 40 ; of various intensity and duration treated at the Department of Dermatology, Medical School in Skopje, from February 2000 to January 2002, were included in this prospective, open-label, randomized, parallel group study. The clinical features of the patients were quantified according to the mean psoriasis area and severity index PASI ; values. Student's t-test for paired samples and two independent samples were used in statistical analysis. Results. The final PASI values were not significantly different for the patients receiving different treatments of guttate psoriasis or generalized psoriasis. The initial PASI values for guttate psoriasis patients treated with betamethasone dipropionate plus UVB with and without penicillin treatment 5.7 2.1 and 5.9 2.5, respectively ; declined to 0.5 0.8 and 1.0 0.9, respectively, after the therapy. The initial PASI values in generalized psoriasis patients receiving PUVA dropped from 24.1 3.6 to 1.7 1.5 by the end of the therapy. Finally, pre-treatment PASI values in patients with generalized psoriasis receiving retinoids decreased from 24.6 3.5 to 0.9 1.1 after treatment. However, patients receiving systemic retinoids for generalized psoriasis had statistically higher incidence of side effects than patients receiving PUVA therapy t 6.458, df 38, p 0.001 ; . Conclusion. Penicillin should be applied in addition to local corticosteroids with UVB in the treatment of guttate psoriasis, since the disease may be triggered by a streptococcal infection. In cases of generalized psoriasis vulgaris, PUVA therapy caused fewer side effects than did systemic retinoids and urecholine.
Just Released Alliance September Issue: Meeting the Millennium Development Goals-- Business As Unusual The September issue of Alliance takes a critical look at the Millennium Development Goals five years on. How much progress has been made? What will be required to meet them by 2015? This issue of Alliance, with guest editor Salil Shetty, director of the UN Millennium Campaign, looks at how companies, foundations, individual philanthropists and social investors can contribute to meeting the MDGs--often in partnership with governments and multilateral agencies. Alliance magazine is published by Allavida, a UKbased international NGO. For more information, visit allavida alliance. Council on Foundations Salary Report and Board Data The salary tables from the 2005 Grantmakers Salary and Benefits Report and the board compensation chapter from the forthcoming Foundation Management Series, Twelfth Edition, Volume III: Foundation Governing Boards and Administrative Expenses in Private Foundations are now available online for Council on Foundations members. The full survey reports will be available later this year. Council members may access salary tables, updated figures for board compensation and reimbursement, and methods used to pay board members by visiting the research section of cof or by clicking here. Call for Nominations Council on Foundations International Committee The Council on Foundations seeks nominations to fill forthcoming vacant positions on its International Committee. The committee seeks members who are committed to its goals of assisting the Council in building and extending the field of international philanthropy and increasing its effectiveness. Self-nominations are welcome. Selections are made with a view toward ensuring that the committee is broadly representative of the Council's membership. U.S. foundations represented on the committee must be Council members. As with all member committees, the International Committee seeks nominees whose participation will ensure diversity across race, gender, foundation type, size and geographic area. The committee's work includes the following.
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Candidates for treatment if they had severe vasoconstricted hypertension that was not responding to aggressive vasodilator therapy or if they had significant renal insufficiency for which ACE inhibitor treatment might be renal protective. Hemodynamic measurements were made before treatment and 2 to 3 days after initiation of therapy. Blood pressure BP ; was measured by using an automated cuff, Accutorr; Datascope, Paramus, NJ ; . Cardiac output was measured by using a previously validated Doppler technique23, 24 UltraCOM Cardiac Output Monitor; Lawrence Medical, Redmond, WA ; . Data were analyzed by using the Wilcoxon signed-rank test. Expected probabilities of adverse outcomes were calculated by using binomial approximation. Mothers and neonates were monitored for complications. Worsening renal function and hyperkalemia were considered maternal complications of ACE inhibitor and bicalutamide.
Sone compared with no antenatal steroid exposure, with a 2-tailed of 5% and a power of 80%. This would provide for the detection of an absolute difference of 4 percentage points in PVL incidence rates between dexamethasone and betam4thasone ie, 6.5% versus 2.5% ; , or an OR of 2.71, with the allowed type I and type II errors. RESULTS From January 1, 2002, to April 30, 2003, a total of 4939 VLBW infants were registered in the network database, 74% of whom received antenatal steroids. Within this group, 3600 VLBW infants met study entry criteria, 635 18% ; of whom received no antenatal steroids, 1227 34% ; of whom received dexamethasone, and 1738 48% ; of whom received betamethasone. Among infants who were exposed to dexamethasone, 445 36% ; received a partial course, 760 62% ; received a complete course, 20 2% ; received multiple courses, and 2 did not have documentation of the number of courses received. Among infants who were exposed to betamethasone, 428 25% ; received a partial course, 1228 71% ; received a complete course, 79 4% ; received multiple courses, and 3 did not have documented course numbers. A total of 2559 71.1% ; infants received at least 1 ophthalmologic examination, and 2947 81.9% ; underwent a cranial sonogram by 36 weeks' postmenstrual age. Baseline Maternal and Neonatal Characteristics Maternal characteristics are shown in Table 1. By preliminary regression modeling adjusting only for center, there were statistically significant differences among the 3 groups with regard to marital status, race, and receipt of prenatal care. Mothers who received antenatal steroids were more likely to be married, be white, and receive at least 1 prenatal care visit compared with those who did not. General neonatal characteristics are shown in Table 2. Adjusting only for network center, preliminary regression analyses revealed statistically significant differences among the 3 groups in several categories. Infants who were exposed to antenatal steroids tended to have lower birth weights and GAs at birth, PROM, intrapartum antibiotics, and higher cord pH compared with those with no antenatal steroid exposure. Postnatally, infants who were exposed to antenatal steroids were less likely to have early-onset sepsis and more likely to be given postnatal steroids for management of chronic lung disease than those who were not exposed to antenatal steroids. There were statistically significant but clinically nonsignificant differences in cesarean delivery rates and Apgar scores among the 3 steroid groups. The sample size for the subgroup of infants who received early indomethacin was too small to perform preliminary modeling, adjusting for network center, among the 3 groups.
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Fective agents for the treatment of bipolar disorder, and as illustrated in the life chart, non-response to one anticonvulsant does not predict non-response to the other thus offering multiple treatment options to the patient struggling with an unstable mood disorder. The patient whose life chart has been presented in this article has resumed a full and productive life. She gave us permission to use her life chart and case history as a message of encouragement and hope: even when there is non-response or refractoriness to some of the available mood stabilizing medications for many years, a series of alternative compounds are now available. Life-charting of the past and present course of bipolar illness will significantly assist in making informed treatment decisions.
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Ever since the landmark observations by Liggins that corticosteroids given to pregnant sheep were associated with accelerated lung maturation in the fetus, ' several studies have shown the beneficial effects, on the fetus, of an antenatal course of corticosteroids for women undergoing premature labour. A Cochrane systemic review indicates that a single antenatal course of corticosteroids reduces the occurrence of respiratory distress syndrome RDS ; , intraventricular haemorrhage IVH ; , and neonatal mortality in babies born prematurely. However, until the US National Institutes of Health Consensus Conference in 1994, physicians were slow to adopt this treatment for women at increased risk of premature delivery. The importance of corticosteroids given antenatally has led to several detailed clinical surveys, which have all identified the spread of the practice of prescribing multiple antenatal courses of corticosteroids when the risk of preterm delivery is jud~ed t? ~1rsist. For example, a recent survey .of obstetrIc UnIts 111 the UK3 showed that 98% of such unItS are prescribing ~epeated courses. This trend is worrying because there is'no good evidence to support an advantage of multiple 'over single courses, 2 whereas there is an increasing body of evidence suggesting that fetal exposure to corticosteroids may have long-term adverse consequences for childhood and adult development. Corticosteroids are important initiators of programmed cell death, so prenatal exposure to these compounds may influence organ growth. In a placebo-conttolled comparison betWeen a single dose and multiple doses of betamethasone in pregnant mice, . a beneficial effect oa neonatal respiratory function and lung histology was noted. However, there was a significant dose-response reduction in fetal-lung weight and ratio of lung weight to bodyweight that persisted into adulthood. A similar study in pregnant rabbits also showed a significant dose-response reduction in fetal birthweight, placental weight, and lung weight.' In pregnant sheep, increasing doses of steroids have a beneficial effect on all aspects of lung function.6 However, there was also a dose-dependent decrease in birthweight and ratio of lung weight to body weight, '." with three courses one week apart resulting in a 25% reduction in birthweight for lambs born preterm and a 19% reduction for those born at term! The effect on growth is not limited to the lung and overall body size. There is compelling evidence of dose-dependent reductions in brain weight and volume, cerebral weight and volume, cerebellar weight, and brain-stem weight in sheep fetuses." Multiple antenatal courses of corticosteroids have also been reported to delay central-nervous-system myelination in sheep fetuses. and to modify neuronal development in the hippocampus and dentate gyrus of rhesus macaque monkeys.'" The effect on neuronal development is probably mediated at the levels of neurogenesis and apoptosis. Because of differences betWeen species in the timing of the brain growth spurt" and in the length of gestation, caution must be applied in the extrapolation of data from one species to another. However, adverse effects in well-controlled animal studies justify a close look for potential adverse effects in human beings. To date there is a paucity of outcome data on the potential adverse effects, on the fetus, of mu1tiple antenatal courses of corticosteroids in human beings. In a recent observational study of 477 preterm singleton infants in Western Austra1ia, 12multiple courses seemed to reduce fetal gro\vthrates and head circumference corrected for sex and.
To evaluate the efficacy of the empirical use of neomycin betamethasone otic solution alone in the treatment of csom-tt during the active stage and bupropion.
Note: For a description of references and other information, refer to the explanation of Committee tables and the accompanying notes at the end of this table. Footnotes: P - Based entirely on projections A - Based in whole or in part on actual data Page 43 of 192.
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Accession number & update 2001-01675-003 R 20070814. Source Comprehensive Psychiatry, Jul-Aug 2001, vol. 42, no. 4, p. 272-282, ISSN: 0010-440X. Publisher: Elsevier Science, Netherlands. Author s ; Langbehn-Douglas-R, Cadoret-Remi-J. Author affiliation Langbehn-Douglas-R, VA Medical Ctr, Dept of Psychiatry, Iowa City, IA, US. Abstract DSM antisocial personality disorder ASPD ; requires retrospective diagnosis of conduct disorder--historical behavior not present in everyone with adult ASPD criteria. Using adoption study data, the authors examined the impact of this requirement on biological and environmental risk associations. The authors also compared clinical correlates of adult antisocial behavior with and without prior conduct disorder. The authors defined 3 subgroups aged 18-47 yrs ; : 30 Diagnostic and Statistical Manual of Mental Disorders-III DSM-III ; ASPD, 25 adult antisocials without conduct disorder, and 142 controls. The authors also examined differences in 2 sociopathy scales and the incidence of co-occurring affective, alcohol, and other substance use disorders. The differences in individual antisocial symptoms was also explored. Having an antisocial biological parent was a specific risk factor for ASPD. Fetal alcohol exposure, male gender, and adverse environment were associated with the adult antisocial syndrome, regardless of conduct disorder history. The 2 antisocial groups were similar with respect to sociopathy scales, co-occurring diagnoses, and the incidence of most individual symptoms. Phenotypic expression for ASPD appears to be manifest before adulthood. Results could not detect clinical important differences between the 2 sociopathic groups. Psyc INFO Database Record c ; 2007 APA, all rights reserved ; . Language English. Publication year 2001.
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Portions during unaffected REM and quiet sleep Schwab et al. 2000b ; . A complete consideration of the usefulness of non-linear and linear methods in quantifying the fetal ECoG would require a comparison of changes in the prediction error based on non-linear analysis with changes of the prediction error based on a linear autoregressive AR ; model. We have previously demonstrated that 24 h of betamethasone exposure results in significant reductions of cerebral blood flow CBF ; in all brain regions but the hippocampus Schwab et al. 2000a ; . Blood flow decreased in the cerebral cortex and in the basal ganglia by about 3540 % and in the thalamus and hindbrain by about 4550 % in comparison to baseline levels. Forty-eight hours after onset of betamethasone infusion, CBF reduction was diminished to about 2530 % by which time CBF was not significantly different from baseline values but still significantly different from that of vehicle-treated fetuses except in the parietotemporal, parietooccipital and occipital cortex. The CBF decrease at 24 h betamethasone exposure would tend to alter the cerebral oxidative metabolism leading to insuffiency of neuronal metabolism since about half of cerebral glucose and oxygen metabolism is used to meet the energy requirements of the ionic pumps that maintain the ionic gradients across cellular membranes Erecinska & Silver, 1989 ; . The first occurrence of altered EEG pattern in the adult brain of humans Trojaborg & Boysen, 1973 ; or cats Hossmann & Schuier, 1980 ; was found at a flow reduction of about 5060 %. Although we demonstrated betamethasone-induced flow reductions of up to the cortex and thalamus, regions essential to ECoG generation, we were unable to demonstrate betamethasone-induced ECoG changes using power spectral analysis. Using non-linear ECoG analysis, however, we were able to detect a distinct reduction of the prediction error of the ECoG in REM sleep during the period of decreased CBF induced by betamethasone infusion. The hindbrain as the region with the most pronounced CBF reduction Schwab et al. 2000a ; plays a substantial role in induction and maintenance of REM sleep Steriade & McCarley, 1990 ; . Moreover, cortical activation during REM sleep requires an increase in and diltiazem.
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