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This is an invitation to attend the 9th International Symposium on Yeasts ISY ; to be held in Sydney on 25-30 August 1996. The ISY is held every four years as an activity of the International Commission on Yeasts ICY ; to foster interest in the science and technology of yeasts. This will be the first time that ISY has been held in Australia. As an added attraction, it will be held concurrently with the 10th International Biotechnology Symposium IBS ; . The ISY and IBS will be held in the heart of Sydney at the spectacular harbourside Sydney Convention Centre. This large purpose-built facility is set in 50 hectares of parks, gardens, museums, shopping malls and amusement areas. It overlooks the harbour with spectacular views of the city. It is a five minute monorail ride to the city centre and within walking distance of several hotels. Sydney itself is the cosmopolitan centre of Australia and is one of the most beautiful cities in the world, with its deepwater harbour, Opera House and sandy beaches. As in previous ISY, the scientific program will be arranged in a number of symposium topics as shown. Leading international researchers will be invited to present the latest advances within the symposia topics. Input into the content of the scientific program has already been obtained from the vast international network of councillors on the International Yeast Commission. In addition, key plenary lectures and poster presentation sessions will complement the symposium papers and there will be opportunity to attend sessions in the IBS program. The Australian Biotechnology Association is honoured to be hosting the symposium. As with any scientific meeting, the success of this ISY will depend on its participants. We hope that you will be able to accept our invitation, and look forward to seeing you in Sydney 1996. Symposium Topics Symposium I: Systematics and Taxonomy. Current status. Classical and molecular approaches. Phylogeny. Basidiomycetes and yeast like organisms. Symposium II: Ecology and Biodiversity. Extreme environments. Soil, Water, Plants, Animals. Symposium III: Biochemistry and Physiology. Metabolism. Stress. Transport. Tolerance of extremes. Symposium IV: Molecular Biology and Genetics. Genome project. Secretion. Expression. Regulation. Symposium V: Cell Cycle. Morphogenesis. Cell interactions. Symposium VI: Ultrastructure. Organelles. Membranes. Cell wall. Symposium VII: Yeasts as Pathogens and Allergens. Epidemiology, Pathogenesis. Treatment. Drug resistance and sensitivity Symposium VIII: Biodegradation. Xylose fermentation. Polysaccharide degradation. Metal accumulation. Symposium IX: Alcoholic Beverages. Brewer's yeast. Wine yeast. Sake yeast. Distillers yeast. Symposium X: Food and Feed Processing. Spoilage yeasts. Yeasts as sources of food additives colours, flavours, vitamins ; . Symposium XI: Enumeration and Detection of Yeasts. Rapid methods. ELISA. Molecular methods. Symposium XII: Fermented Foods. Baker's yeast. Traditional fermented foods. Yeasts in dairy products Symposium XIII: Biocontrol and Probiotic Yeasts. Biocontrol of food spoilage. Probiotic species in human and animal foods. Symposium XIV: Fermentation Technology. Immobilised yeasts. Novel bioreactor systems Symposium XV: Training and Education in Yeast Biology and Technology. Scientific Organising Committee. Desmond Clark-Walker, Australian National University; Ian Dawes, University of New South Wales; David Ellis, Adelaide Childrens Hospital; Graham Fleet, University of New South Wales; Phillip Franks, Burns Philp, Food and Fermentation Division; Paul Henschke, Australian Wine Research Institute; Ian Jenson, Burns Philp, Food and Fermentation Division; Neville Pamment, University of Melbourne; Ken Watson, University of New England. The First Circular on the 9th IYS Sydney has been sent out. For further information contact.
The quality of trial 039 SmithKline Beecham ; could be easily assessed using information obtained from several study publications see Table 4 ; .5456 Details of trial 30-49 Schering-Plough Ltd. ; were only published in abstract form and thus a full assessment of study quality relied on the use of confidential information contained within the company submission see Table 4 ; .59 Trial 039 used a centralised telephone method to randomly assign 235 participants to the two study groups and was, therefore, truly randomised. Participants were stratified according to Ptsensitivity, age and ascites at baseline. Also implicit in this method of randomisation is the adequate concealment of the allocation procedure, which avoids the possibility of tampering and thus bias in treatment allocation. All of those patients originally included in the randomisation process were accounted for and the results of the trial were presented in terms of an intention-to-treat ITT ; analysis as well as a per protocol analysis. However, the ITT analysis used was not a true one because five patients from the topotecan group and four from the paclitaxel group who were included in the randomisation procedure were not included in the ITT analysis because they never received the treatment to which they were assigned. Of those assigned to receive topotecan, three participants subsequently refused treatment and withdrew their consent and two died one from progressive disease and the other from pulmonary embolism ; . Three of the participants assigned to the paclitaxel group subsequently withdrew their consent and refused treatment and one had a performance status of 4 and thus did not fulfil the inclusion criteria for the trial. Not including these participants in the ITT analysis could introduce bias because these patients could have had a poorer prognosis which was certainly true for the participant who died in the topotecan group ; . However, further trial withdrawals were included in the ITT analysis and reasons for their withdrawal were clearly stated. The topotecan and paclitaxel study groups were similar at baseline in terms of the six characteristics stated in the quality checklist identified by the external review panel ; . These factors were chosen for their potential importance in predicting disease progression and treatment response and included treatment-free interval, disease bulk, number of previous regimens, age, histology and performance status. The criteria used to select patients for inclusion in the trial were also stated, and appeared to be reasonable and, for example, naproxyn.

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HOSPITAL BENCHMARKING PERFORMANCE IMPROVEMENT LINKS Links to these sources are provided solely as a service by the Massachusetts Board of Registration in Medicine. These links do not constitute endorsement of these studies by the Board and none should be inferred. Medline : entrez query.fcgi?db PubMed ; * Obtain journal articles from your librarian for complete information. Years. No double vision or other visual abnormalities were noted. Medication consisted of an over-thecounter inhaler. An examination revealed mild left proptosis and slight hypoglobus. An intranasal examination revealed polyps filling both nasal cavities with extension into the nasal vestibule. The remaining physical examination findings were normal. A coronal CT scan of the head, without contrast, re and propecia. General practice nursing is one of the youngest, fastest growing and broadest areas of nursing. This makes it an exciting branch of the profession to join, and increasing numbers of newly qualified nurses are showing an interest in a career in general practice. GPNs are a group with strong attributes. They have a broad spectrum of nursing skills and a flexible approach to providing care. This is backed up with good negotiating skills, developed through their close working relationship with GPs. In addition, they have excellent assessment and problem-solving skills, and a holistic approach to providing supportive, patient-centred care. Many of these skills have been acquired as a result of the historical development of practice nursing and the challenges that GPNs have been presented with. For a number of years there has been a political focus on primary care. Reflecting this, practice nurses have become adept at absorbing political changes into their work, often in response to a new white paper outlining the latest health strategy. These challenges have enabled them to face the future with openness and optimism, combined with a spirit of collaboration. In an ever-changing environment, the educational and professional development needs of GPNs are hard to keep up with. Despite this, practice nurses have a tradition of training for particular clinical functions as dictated by the demands of their work. However, the breadth of their clinical duties, combined with difficulties in getting time away from the practice, make it quite a challenge to maintain competence and keep up to date. There has been political support for the expansion of practice nurses' roles for many years. Responding to this, GPNs have taken on more delegated work from GPs and increased their scope of practice considerably. The new NHS: modern, dependable 1997 ; began the Westminster government's current health reforms, and included devolving decisionmaking and financial management to local level, with clinicians including GPNs ; working at executive board level. Likewise, in the wake of devolved government in Scotland and in response to Delivering for Health 2005 ; there has been an increased focus on delivery of care in the community, with corresponding challenges and opportunities for GPNs. The effect on practice nurses of this process of delegation has been to give them direct opportunities to shape and influence the provision of care. It has also provided the potential for career progression as a PCT nurse. New areas of practice now available to GPNs include diagnostic testing, making referrals, prescribing, performing triage, minor surgery and outpatient procedures. Across the UK, the new GMS contract has created further opportunities for extended roles and greater responsibilities Chatterjee, 2004 ; , including nurse partnerships. This expansion and diversification of what GPNs do has resulted in bigger teams with a wider range of skills. With these increased responsibilities it has become clear that there is a greater need than ever for the skills of GPNs to be supported. This is particularly important as the independent contractor status of GPs means that uniformity of training and supervision for GPNs is difficult to achieve.
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Rescription strength form aka brand name naprosyn, generic naproxen ; they are 500mg tablet taken 2-3 times a day and tenormin. Acetaminophen up to 1g QID ; has been compared to an NSAID in patients with OA in 3 clinical trials. In each of the studies, the NSAID was not more effective in achieving pain relief or decreasing joint stiffness than acetaminophen, even in patients who had signs and symptoms of joint inflammation. Many patients with OA may not need the `anti-inflammatory' effects of an NSAID. Comparative Safety The most common side effects of the NSAIDs involve the gastrointestinal tract - GI intolerance abdominal pain, dyspepsia ; and GI ulceration. GI intolerance is dose-related. In general, the proprionic acid derivatives ibuprofen, naproxen, tiaprofenic acid, ketoprofen ; are less likely to cause stomach upset than other NSAIDs. Enteric coating has significantly improved the GI tolerability of some NSAIDs e.g. ASA ; . However, enteric coating of well tolerated NSAIDs e.g. naproxen; Naprosyn-E ; offers minimal improvement over uncoated naproxen. While some studies show that enteric coated naproxen is better tolerated usually at higher doses ; , other studies indicate no difference in tolerability between enteric coated and uncoated naproxen. Enteric coating does not reduce the incidence of NSAID-induced ulcers. GI ulceration and the resultant complications perforation, hemorrhage ; are the more serious GI side effects associated with NSAIDs. All routes of administration oral, rectal and parenteral ; can cause damage to the GI mucosa. The use of `prophylactic' medications to prevent NSAID-induced ulcers remains controversial. Misoprostol Cytotec ; is the only drug proven to decrease the incidence of NSAID-induced gastric and duodenal ulcers. H2-antagonists.

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FM O'Prey1, 2, SA Wright1, DJ Rea1, 3, C McGivern3, RD Plumb1, 2 , W Henry2, GD Johnston1, 2, GE McVeigh1, 2 1. Dept of Therapeutics and Clinical Pharmacology, Queens University Belfast 2. Belfast City Hospital 3. NI Regional Medical Physics Agency, Royal Victoria Hospital, Belfast.
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Gross Toxicity All drug-treated mice survived the 10-week administration period with no signs of gross toxicity such as failure to groom or respond normally to handling. However, body weight gain of drug treated mice was slightly affected by drug treatment and is shown in Table 1. Drug administration was associated with a trend toward reduced body weight compared to control mice throughout the evaluation period. There was an initial retardation of growth in mice receiving TX and DX compared to untreated controls in the first two weeks p 0.05 ; . Subsequently, between weeks 2 and 10 the final observation point ; TX- and DX-treated mice gained approximately 10% in body weight, such that at week 10 there was no statistically significant difference in body weight between drug-treated groups and controls. Peripheral Blood Indices Changes in peripheral blood indices were evident at week 2, particularly in mice administered HU. The WBC count in animals receiving HU was 10-fold higher than control mice p 0.05 ; and was elevated in all subsequent evaluations Fig. 1A ; . The peak WBC was reached at week 8, being 15-fold higher than the untreated group, subsequently declining to approximately threefold higher than normal at week 10. It is evident from the WBC differential Fig. 2A ; that the majority of these peripheral white cells were lymphocytes. Compared to HU, the effect of TX and DX on the WBC was more modest Fig. 1A ; . DX did elevate the WBC slightly, with the only meaningful increase occurring at week 8. However, this increase was still only one-fourth of the HU-treated group level. The other time points were similar to that of the control group. The WBC of mice treated with TX was the same as controls or slightly lower throughout the duration of the experiment, with negligible changes in the white cell differential count Fig. 2 ; . Furthermore, HU also influenced the numbers of circulating neutrophils, with elevations evident at all time points, particularly week 8 where a several-fold increase was observed Fig. 2B.

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