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CIGNA Behavioral Health cannot provide you with legal advise on the use of any release form for your practice. The following is a sample only. You should obtain the advice of legal counsel for your practice. ; Consent for Release of Confidential Information to Primary Care Physician I, hereby authorize Participant's Name ; Practitioner's Name ; to disclose to my Primary Care Physician, all the clinical information about me as may be necessary to permit my Primary Care Physician to monitor the continuity of my care and to inform my Primary Care Physician of my health status. This authorization becomes effective , and may be revoked by me in writing at any time, with the exception of any actions already taken to coordinate my care. Unless earlier revoked by me, this authorization automatically terminated the earlier of six 6 ; months from the effective date. I understand that this authorization does not extend to the release of any AIDS HIV information unless I also placed my initials here . I further understand that the information authorized by this release will be released to the authorized representative only, for purposes noted above. I understand I or my legal representative ; entitled to a copy of this authorization form for my records, for instance, acenocoumarol sintrom.
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Pryor DB, Shaw L, McCants CB, et al. Value of the history and physical in identifying patients at increased risk for coronary artery disease. Ann Intern Med 1993; 118: 81-90. Chaitman BR, Bourassa MG, Davis K, et al.Angiographic pre valence of high-risk coronary artery disease in patient subsets CASS ; .Circulation 1981; 64: 360-7. Pryor DB, Harrell FEJ, Lee KL, Califf RM, Rosati RA timating the likelihood of significant coronary artery disease. J Med 1983; 75: 771-80. Braunwald E, Mark DB, Jones RH, et al. Unstable angina: diagnosis and management. AHCPR Publication No 94-0602. Braunwald E, Califf RM, Cannon CP, Fox KAA, Fuster V, Gibler W B, Harrington RA, King SB, Kleiman N, Theroux P, Topol EJ, Van de Werf F, White HD, Willerson JT. Redefining Medical Treatment in the Management of Unstable Angina.Am J Med 2000; 108: 41-53. The PURSUIT Investigators. Inhibition of glycoprotein IIb IIIa with eptifibatide in patients with acute coronary syndromes. N Engl J Med 1998; 339: 436-443. Adams JE, Abendschein DR, Jaffe AS. Biochemical markers of myocardial injury: is MB creatinine kinase the choice for the 1990s? Circulation 1993; 88: 750-63. Mair J, Morandell D, Genser N, Lechleitner P, Dienstl F, Puschendorf B. Equivalent early sensitivities of myoglobin, creatinine kinase MB mass, creatinine kinase isoform ratios, and cardiac troponins I and T for acute myocardial infarction.Clin Chem 1995; 41: 1226-72. Van de Werf F. Cardiac troponins in acute coronary syndromes. NEJM.1996; 335: 1388-1389. Ohman EM, Armstrong PW, Christenson RH, et al. Cardiac troponin T levels for risk stratification in acute myocardial ischaemia.NEJM.1996; 335: 1333-1341. Lindahl B, Venge P, Wallentin L. Relation between troponin T and the risk of subsequent cardiac events in unstable coronary artery disease. Circulation.1996; 93: 1651-1657. Antman EM, Tanasijevic MJ, Thompson B, et al rdiac-specific troponin I levels to predict the risk of mortality in patients with acute coronary syndromes. NEJM.1996; 335: 1342-1349. Stubbs P, Collinson P, Moseley D, et al. Prognostic significance of admission troponin T concentrations in patients with myocardial infarction.Circulation 1996; 94: 1291-97. Newby LK, Christenson RH, Ohman EM, et al. Value of serial troponin T measures for early and late risk stratification in patients with acute coronary syndromes. Circulation. 1998; 98: 1853-1859. Christenson RH, Duh SH, Newby LK, et al rdiac troponin T, and cardiac troponin I: relative values in short-term risk stratification of patients with acute coronary syndromes. Clin Chem. 1998; 44: 494-501. Olatidoye AG, Wu AH, Feng YJ, Waters D. Prognostic role of troponin T versus troponin I in unstable angina pectoris for cardiac events with meta-analysis comparing published studies. J Cardiol.1998; 81: 1405-1410. Effects of tissue plasminogen activation and a comparison of early in vasive and conservative strategies in unstable angina and non-Q wave m yocardial infarction: results of the TIMI IIIB trial.Thrombolysis In Myocardial Ischaemia.Circulation 1994; 89: 1545-56 Zimmerman J, Fromm R, Meyer D, et al. Diagnostic Markers Cooperative Study for the diagnosis of myocardial infarction.Circulation 1999; 99: 1671-7. A Report of the American College of Cardiology American Heart Association Task Force on Practice Guidelines Committee on the Management of Patients with Unstable Angina ; ACC AHA Guidelines for the Management of Patients with Unstable Angina and Non-STsegment Elevation Myocardial Infarction JACC Vol.36, 2000: 970-1062 Management of acute coronary syndromes without persistent ST-segment elevation Recommendations of the Task Force of the European Society of Cardiology European Heart Journal 2000 ; 21, 1406-1432.
Investigational New Drug Application, or IND: An application that a drug sponsor must submit to FDA before beginning tests of a new drug on humans. The IND contains the plan for the study and is supposed to give a complete picture of the drug, including its structural formula, animal test results, and manufacturing information. New Drug: A drug first investigated or proposed for marketing after 1938--that is, a drug that was not generally recognized as safe and effective before that date. New Drug Application, or NDA: An application requesting FDA approval to market a new drug for human use in interstate commerce. The application must contain, among other things, data from specific technical viewpoints for CDER review-- including chemistry, pharmacology, medical, biopharmaceutics, statistics and, for anti-infectives, microbiology. New Molecular Entity, or NME: A compound that can be patented, which has not been previously approved. Parallel Track Mechanism: Policy that makes promising investigational drugs for AIDS and other HIV-related diseases more widely available under "parallel track" protocols, while the controlled clinical trials essential to establish the safety and effectiveness of new drugs are conducted. The system established by the policy is designed to make the drugs more widely available to patients with these illnesses who have no therapeutic alternatives and cannot participate in the controlled clinical trails, for example, trombosebeen.
Three coumarin-derived drugs are currently used therapeutically as anti-coagulants. These do not differ in their effect on vitamin K metabolism, but are different in terms of pharmacokinetics. The elimination half-life of warfarin is 4070 h, whereas those of acenocoumarol and phenprocoumon are 3-10 h and 90140 h respectively. These differences have implications in patient management: for example phenprocoumon takes over 2 weeks to reach steady-state, whereas acenocoumarol has to be administered on a twice daily basis4.
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Although the precise mechanism of the interaction between methylprednisolone and oral anticoagulants is unclear, several lines of evidence strongly suggest that it is due to inhibition of oral anticoagulant catabolism by high-dose methylprednisolone. First, the increase in the INR observed after methylprednisolone administration occurred through a vitamin K dependent pathway. The INR increased regardless of the anticoagulant used acenocoumarol, warfarin, or fluindione, a noncoumarin indanedione anticoagulant that is common in Europe ; . In addition, this increase was rapidly reversed by administration of vitamin K 14 ; . Second, because oral anticoagulants are almost completely absorbed from the gastrointestinal tract 9 ; , it is unlikely that elevated fluindione concentrations were due to increased absorption. Third, because we measured total fluindione, it is unlikely that the elevated concentrations resulted from a change in the ratio between free and protein-bound fluindione. Fourth, methylprednisolone inhibits the cytochrome P450 enzyme system 17 ; , which is involved in the metabolism of oral anticoagulants 18 ; . It therefore and acetylsalicylic.
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Mean SD ; * Least squares Mean SD ; * mean SE ; c Daily servings of fruit 2.8 2.1 ; Daily servings of vegetables 4.0 2.7 ; Daily servings of fruits and vegetables 6.8 3.9 ; Total daily caloric intake 1871 652 ; Percent calories from total fat 29 7 ; Daily dietary fiber g ; 23.1 9.5 ; 2.6 4.0 6.6 ; 0.2 ; 0.2 ; 41 ; 0.4 ; 0.6 ; 2.7 3.9 6.6 ; 2.6 ; 3.9 ; 696 ; 6 ; 9.4 and salbutamol, for instance, diclofenac.
Since the "no drug" group behaved the same as the experimental groups during the control and shock periods, data are presented as pooled data for the control and shock periods Tables I and III ; . The "no drug" group also behaved the same as a larger control group previously reported.2 As blood was removed mean arterial pressure M.A.P. ; , central venous pressure C.V.P. ; , pulmonary artery pressure P.A. ; , left atrial L.A. ; pressure and cardiac output Q ; fell Figure 1 and Table I ; . Between the early and late periods of hypotension, however, pulmonary artery pressure PA ; rose significantly 7 2 mm Hg, p 0.025 ; while left atrial pressure fell 1.4 0.5 mm Hg, p 0.025 ; Figure 1 ; , CVP fell 0.8 0.3 cm H2O, p 0.01 ; and mean arterial pressure did not change early shock 54 3 mm and late shock 52 1 mm Between the early and late hypotensive period the mean pulmonary artery to left atrial pressure gradient rose 9.5 1.7 mm Hg p 0.005 ; Figure 1 ; . This, along with a slight fall in Q 0.26 0.08 1 min, p 0.01 ; , resulted in a 74.9 11.9 per cent increase in PVR p 0.001, Figures 1 and 2 ; . During the hypotensive period pH fell 0.099 0.021, p 0.001 ; due to a metabolic acidosis as B.E. fell 7.17 0.79 mEq 1, p 0.001 ; . Pa 02 rose 13.5 1.9 mm Hg, p 0.001 ; as the.
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Associations of Fibrinogen, Factor VII and PAI-1 with Baseline Findings among 10, 500 Male Participants in a Prospective Study of Myocardial Infarction The Prime Study P.-Y. Scarabin, M.-F. Aillaud, P. Amouyel, A. Evans, G. Luc, J. Ferrires, D. Arveiler, I. Juhan-Vague 749 FVIII Coagulant Activity and Antigen in Subjects with Ischaemic Heart Disease G. I. Rice, P. J. Grant 757 Factor V Leiden and Prothrombin Gene G 20210 A Variant in Children with Ischemic Stroke W. Zenz, Z. Bod, J. Plotho, W. Streif, Ch. Male, G. Bernert, L. Rauter, G. Ebetsberger, K. Kaltenbrunner, P. Kurnik, A. Lischka, F. Paky, R. Ploier, G. Hfler, C. Mannhalter, W. Muntean 763 Fibrin Deposition in Squamous Cell Carcinomas of the Larynx and Hypopharynx H. Brdos, A. Juhsz, G. Rpssy, R. Adny 767 Prospective, Randomised Trial of Two Doses of rFVIIa NovoSeven ; in Haemophilia Patients with Inhibitors Undergoing Surgery A. D. Shapiro, G. S. Gilchrist, W. K. Hoots, H. A. Cooper, D. A. Gastineau 773 French Previously Untreated Patients with Severe Hemophilia A after Exposure to Recombinant Factor VIII: Incidence of Inhibitor and Evaluation of Immune Tolerance C. Rothschild, Y. Laurian, E. P. Satre, A. Borel Derlon, H. Chambost, P. Moreau, J. Goudemand, A. Parquet, J. Peynet, M. Vicariot, P. Beurrier, S. Claeyssens, A. Durin, A. Faradji, E. Fressinaud, S. Gaillard, V. Gurin, C. Gurois, G. Pernod, P. Pouzol, J. F. Schved, G. Gazengel 779 Deletion Polymorphism in the Angiotensin-converting Enzyme Gene as a Thrombophilic Risk Factor after Hip Arthroplasty C. S. Philipp, A. Dilley, P. Saidi, B. Evatt, H. Austin, J. Zawadsky, D. Harwood, D. Ellingsen, E. Barnhart, D. J. Phillips, W. C. Hooper 869 Hyperhomocysteinemia and Venous Thrombosis: A Meta-Analysis M. den Heijer, F. R. Rosendaal, H. J. Blom, W. B. J. Gerrits, G. M. J. Bos 874 No Association between the 20210 G A Prothrombin Gene Mutation and Premature Coronary Artery Disease J. W. Eikelboom, R. I. Baker, R. Parsons, R. R. Taylor, F. M. van Bockxmeer 878 Augmented Platelet Aggregation as Predictor of Reocclusion after Thrombolysis in Acute Myocardial Infarction T. K. Nordt, M. Moser, B. Kohler, J. Ruef, K. Peter, W. Kbler, C. Bode 881 A Cost-effectiveness Analysis of Aspirin versus Oral Anticoagulants after Acute Myocardial Infarction in Italy - Equivalence of Costs as a Possible Case for Oral Anticoagulants J. Gianetti, G. Gensini, R. De Caterina 887 Tissue Factor Expression and Metastatic Potential of Colorectal Cancer C. Shigemori, H. Wada, K. Matsumoto, H. Shiku, S. Nakamura, H. Suzuki 894 Warfarin or Acenocoumarol: Which Is better in the Management of Oral Anticoagulants D. Barcellona, M. L. Vannini, L. Fenu, C. Balestrieri, F. Marongiu 899 Contribution of Acute-Phase Proteins and Cardiovascular Risk Factors to Erythrocyte Aggregation in Normolipidemic and Hyperlipidemic Individuals X. Weng, G. O. Roederer, R. Beaulieu, G. Cloutier 903 Retrospective Neuropathological Review of Prion Disease in UK Haemophilic Patients C. A. Lee, J. W. Ironside, J. E. Bell, P. Giangrande, C. Ludlam, M. M. Esiri, J. E. McLaughlin 909.
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Venous thrombosis all had relative risks that included 1, indicating no significant difference between treatments. Most information was for mortality, where there were consistent results in 13 trials Figure 1 ; . For this outcome it was possible to perform sensitivity analyses. No sensitivity analysis produced any different result, using only those trials unequivocally properly randomised, or including those where the method of randomisation was not stated, or trials where the shorter period of bed rest was less than seven days, or those where anticoagulants or thrombolysis was used Table 2 and alpha-lipoic.
A number of changes to the naming of medicines in this country are to be made, in order to bring Britain into line w ith other countries. The current British Approved Names BANs ; are to be replaced with recommended International NonProprietary Names rINNs ; . As of 1st December 2003, where the names differ, the rINN has been the correct name. How ever, prescribers and dispensers are asked, where BANs and rINNs differ, to use rINNs by 30th June 2004. Whilst many names have not changed and a majority of changes are minor, some names are signific antly different with the risk of patient and professional confusion, and medication error. For the full list of changes, please consult the Medicines and Healthcare products Regulatory Agency website: mhra.gov . How ever, the follow ing medicines have been considered to have the most signific ant change: FORMER BAN Dothiepin Benzhexol Thyroxine Trimeprazine Dicyclomine Sodium Ironedetate Methotrimeprazine Amethocaine Colistin sulphomethate sodium Cysteamine Gestronol Hexamine Hydroxyurea Nicoumalone Quinalbarbitone Salcatonin Stilboestrol Thymoxamine Tribavirin NEW BAN Dosulepin Trihexyphenidyl Levothyroxine Alimemazine Dicycloverine Sodium Feredetate Levomepromazine Tetracaine Colistimethate sodium Mercaptapine Gestonorone Methenam ine Hydroxy Carbam ide Acenocomarol Secobarbital Calcitonin Diethylstilbestrol Moxisylyte Ribaviron.
Reprint requests to: Margaret E. Black, Department of Pharmaceutical Sciences, P.O. Box 646534, Washington State University, Pullman, WA 99164-6534, USA; e-mail: blackm mail u ; fax: 509 ; 335-5902. Article and publication are at : proteinscience cgi doi 10.1110 ps.2460102 and amantadine.
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Acenocoumarol. B ; , warfarin. C ; , phenprocoumon. D ; , p-chlorowarfarin as the IS. For accenocoumarol A ; and warfarin B ; , only background noise was observed. Phenprocoumon C ; was unambiguously identified and amiloride.
Articles marked with an asterix * ; relate to work described in this thesis. Crohbach MJJS, Toorenburg-Beijer B, van der Wal J, van Leeuwen JT, van Puijenbroek EP, van der Laan JR, Geijer RMM. NHG-standaard allergische en hyperreactieve rhinitis. Huisarts en Wetenschap 1995; 38: 216-17. Van Puijenbroek EP, Meyboom RHB. Slaapstoornissen tijdens het gebruik van mefloquine. Pharmaceutisch weekbl 1995; 130: 477-9. Van Puijenbroek EP. Stemklachten als bijwerking van geneesmiddelen. Pharmaceutisch Weekbl 1996; 131: 551-4. Van Puijenbroek EP, Van Amerongen CA. Is het eerste-uitgifte signaal van nut voor postmarketing surveillance? Resultaten van een pilot study. Pharmaceutisch Weekbl 1996; 16: 459-62. Van Puijenbroek EP, Du Buf-Vereijken PW, Spooren PF, van Doormaal JJ. Possible increased risk of rhabdomyolysis during concomitant use of simvastatin and gemfibrozil. J Intern Med 1996; 240: 403-4. Van Puijenbroek EP, Egberts ACG, Krom HJ. Visual hallucinations and amnesia associated with the use of zolpidem. Int J Clin Pharmacol Ther 1996; 34: 318. Meyboom RHB, Verduijn MM, Steenvoorden MG, Dekens-Konter JA, van Puijenbroek EP. Reversibele tandverkleuring tijdens oraal gebruik van antibiotica. Ned Tijdschr Geneeskd 1996; 140: 207-9. Van Puijenbroek EP, Egberts ACG, Trooster JF, Zomerdijk J. Reduction of migrainous headaches during the use of acenocoumarol. Headache 1996; 36: 48. Meyboom RHB, van Puijenbroek EP, Vinks MHAM, Lastdrager CJ. Disturbance of withdrawal bleeding during concomitant use of itraconazole and oral contraceptives. N Z Med J 1997; 110: 300. Van Puijenbroek EP, van Grootheest AC. Bijwerkingen, een verantwoordelijkheid van artsen? Medisch Contact 1997; 52: 1156-8. In 't Veld BA, van Puijenbroek EP, Stricker BHCh. Overgevoeligheidsracties bij gebruik van mebeverine. Ned Tijdschr Geneeskd 1997; 141: 1392-5. Bouvy M, Gerts BMF, Smakman-Nossbaum E, van Puijenbroek EP. Fixed drug eruption bij gebruik van norfloxacine. Huisarts en Wetenschap 1997; 40: 595-7.
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