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Ties of going out of bed, and change in mood. Conclusion : These preliminary results suggest that sleep restriction is effective when used alone. In addition, it indicates that the treatment response and duration may differ among patients. Attention to common FAQ may aid effective home implementation. Further analyses are needed to evaluate biological and psychological mechanisms related to sleep improvements. Support optional ; : Research supported by CIHR and poor sleep, we extracted sleep equivalent groups from non-complaining poor sleepers NPS ; and people with insomnia PWI ; . Methods : A random digit dialing survey collected for 14 days of sleep diaries and measures of health and daytime functioning from 772 participants. The sleep diaries provided the following measures: total sleep time TST ; , sleep onset latency SOL ; , wake time after sleep onset WASO ; , number of nighttime awakenings, sleep efficiency, time spent napping and perceived sleep quality. Participants also completed the following daytime functioning measures: the Insomnia Impact Scale IIS ; , Epworth Sleepiness Scale, Fatigue Severity Scale, State-Trait Anxiety Inventory STAI ; , and Beck Depression Inventory BDI ; . Participants who exhibited mean SOL or WASO 31 minutes were classified as poor sleepers. Results : Criteria that matched subjects on sleep variables were empirically derived, so that daytime functioning could be compared in equivalent groups of PWI and NPS. Individuals who reported cancer were excluded from the analysis because the groups had a significant difference in cancer rates. The results revealed that PWI N 16 ; and NCI N 21 ; had significant differences in BDI [t 35 ; 2.04, p .049], M 13.5, SD 8.6 for PWI and M 8.3, SD 6.2 for NCI; STAI [t 35 ; 3.59, p .001], M 45.0, SD 10.5 for PWI and M 34.4, SD 7.5 for NCI; and IIS [t 35 ; 2.24, p .031], M 116.6, SD 27.4 for PWI and M 98.0, SD 23.1 for NCI. Conclusion : Two groups were formed based on presence or absence of an insomnia complaint from a sample of people who satisfy the sleep detriment criteria for insomnia. Groups were then matched based on sleep using empirically derived criteria. People who complained of insomnia reported more impaired daytime functioning. Possible explanatory factors to account for differential daytime impairment are lower sensitivity to sleep detriments among NPS or lesser propensity to emotional difficulties among NPS. Support optional ; : This Research was supported by the National Institute on Aging grants AG12136 and AG14738.
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[1] Bender BG, Lerner JA, Poland JE. Association between corticosteroids and psychologic change in hospitalized asthmatic children. Ann Allergy 1991; 66 5 ; : 4149. [2] Bodnoff SR, Humphreys AG, Lehman JC, Diamond DM, Rose GM, Meaney MJ. Enduring effects of chronic corticosterone treatment on spatial learning, synaptic plasticity, and hippocampal neuropathology in young and mid-aged rats. J Neurosci 1995; 15 1 Pt 1 ; 619. [3] Bonne O, Brandes D, Gilboa A, Gomori JM, Shenton ME, Pitman RK, et al. Longitudinal MRI study of hippocampal volume in trauma survivors with PTSD. J Psychiatry 2001; 158 8 ; : 124851. [4] Brambilla P, Harenski K, Nicoletti MA, Mallinger AG, Frank E, Kupfer DJ, et al. Anatomical MRI study of basal ganglia in bipolar disorder patients. Psychiatry Res 2001; 106 2 ; : 6580. [5] Bremner JD, Narayan M, Anderson ER, Staib LH, Miller HL, Charney DS. Hippocampal volume reduction in major depression. J Psychiatry 2000; 157 1 ; : 1158. [6] Brown ES, Woolston J, Frol A, Bobadilla L, Khan DA, Hanczyc M, et al. Hippocampal volume, spectroscopy, cognition, and mood in patients receiving corticosteroid therapy. Biol Psychiatry 2004; 55 5 ; : 53845. [7] Brunson KL, Eghbal-Ahmadi M, Bender R, Chen Y, Baram TZ. Long-term, progressive hippocampal cell loss and dysfunction induced by early-life administration of corticotropin-releasing hormone reproduce the effects of early-life stress. Proc Natl Acad Sci USA 2001; 98 15 ; : 885661. [8] De Bellis MD, Hall J, Boring AM, Frustaci K, Moritz G. A pilot longitudinal study of hippocampal volumes in pediatric maltreatmentrelated posttraumatic stress disorder. Biol Psychiatry 2001; 50 4 ; : 3059. [9] Frodl T, Meisenzahl EM, Zetzsche T, Hohne T, Banac S, Schorr C, et al. Hippocampal and amygdala changes in patients with major depressive disorder and healthy controls during a 1-year follow-up. J Clin Psychiatry 2004; 65 4 ; : 4929. [10] Keshavan MS, Bagwell WW, Haas GL, Sweeney JA, Schooler NR, Pettegrew JW. Changes in caudate volume with neuroleptic treatment. Lancet 1994; 344 8934 ; : 1434. [11] Kirschbaum C, Wolf OT, May M, Wippich W, Hellhammer DH. Stress- and treatment-induced elevations of cortisol levels associated with impaired declarative memory in healthy adults. Life Sci 1996; 58 17 ; : 147583 and accolate, for example, symptom for zyrtec.
Higher in the patient group. This is not surprising, as all vasoactivemedications had been withdrawn, and six of the patients were currently being treated for hypertension. The greater body mass index BMI ; is also in keeping with obesity as a risk factor for CAD 41 ; . No correlation existed between weight or BMI and Salb-induced AIx response. The lower resting AIx and HR in the control subjectsare consistent with a greater degree of cardiovascular fitness. Previous studies have shown that thesefactors are associated with changesin aerobic capacity 13 ; . It possible that the lower GTN response in the CAD patients reflected impaired smooth muscle dilation. Over the entire clinical validation cohort n 22 ; , a significant relationship existed between the Salb response and the GTN response r 0.62, p 0.005 ; . Within either the controls or the CAD patient groups in isolation, no such relationship was evident. Such a relationship has been previously demonstrated in a meta-analysis of studies of 800 subjects from a single laboratory using flow-mediated dilation 35 ; . Study limitations. In these pilot studies, the study groups were small in number. This, and the smaller effect of Salb compared to GTN, resulted in the nonsignificant two-way ANOV A results. To avoid complicating effects of endogenous hormones, only males were studied. Therefore, this work needsto be extended to examine the effectsof Salb on the AIx in women. Although the reproducibility of this technique was very good in the current study, reproducibility may need to be further assessed an older population, in one more representativeof target study populations. Further trials in vanous disease and risk-factor categories hypercholesterolemia, diabetes, hypertension, smoking ; are neededto determine whether the technique will be sensitive to isolated risk-factor analysis. In view of the recently recognized effect of beta2-receptor polymorphisms on resting and exercise BP responses to beta2-agonist infusions 42, 43 ; , additional studies are required to examine the effect of common polymorphisms on the inhaled Salb AIx response, both in healthy and in diseasedpopulations. Conclusions. In this study we have shown that the peripheral arterial pressurewaveform is sensitiveto beta2-receptor stimulation by low-dose inhaled Salb. These changesappear to be related to NO release; the technique is sufficiently sensitive to detect differencesbetween small clinical subject groups and are reproducible over the short term. Arterial waveform changesfollowing Salb may provide a noninvasive method of measuring "global" arterial endothelial function, and further studies are needed to document effectsin larger patient groups and to examine the effect of therapies. The portability and simplicity of the technique make large-scale population studies possible.
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Time course and concentration-effect relationship of procainamide-induced vacuolization of rabbit SMCs. The 250 M concentration of procainamide did not induce vacuolization over the whole 48 h observation period, but the 1 mM level has an effect on many cells fig. 3 ; . Omission of FBS from the culture medium did not change importantly this concentration-effect relationship data not shown ; . Vacuolization is detectable as soon as 1 h after 2.5 mM drug application and continues to progress over 24 h, but without apparent mortality or detachment over 48 h. Prolonged 4 h ; exposure to the drug at 2.5 mM changed the morphology of the cells, with a retraction of the pseudopodia. Vacuolization induced by procainamide-related drugs was slowly reversed by drug washout with fresh culture medium fig. 3 ; . A wound closure assay showed that the 250 M and 2.5 mM concentrations of procainamide are equally effective to reduce and actonel.
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Domestic production According to Goskomstat, in July 2007 the output of domestic pharmaceutical companies was $177 Mln., the growth in dollar prices accounted for 30% compared to the same figure a year ago. The value of drugs produced by domestic enterprises in July of 2007 decreased 5% compared to the respective figure in June of 2007. The top 10 producers by output value in July 2007 are listed in Table 1. The total output of these leaders accounted for $97.8 Mln., which is 55% of the total pharmaceutical production by value and aldactone.
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Considering that drug administration at a national level covered all the municipalities in 1995, that some of the categories of sheep remain as reservoirs up to the age of 7 or years, and E. granulosus eggs may survive in the environment up to one year, it is necessary to maintain the attack phase by monthly praziquantel treatment of dogs in a guided manner until 2003-2004. Some municipalities may enter the consolidation phase before that date. This phase may be reached after achieving a low re-infection potential in adult sheep under 1.
Whole-cell 59 with dried cells 54 with immobilized cells 55 with permeabilized cells 54 with purified enzymes 56 with spores 54 with vegetative or washed cells 54 Bis glycinato ; copper II ; hydrate 257 Biuret method for protein determination 525 Black-box model 96 Blakeslea trispora 117 Blasticidin [2079-00-7] 183 Blood coagulation factors 480 Blood coagulation factor XII Hageman factor ; 365 Blotting methods in genetic engineering northern blotting 466 southern blotting 465 western blotting for identifying specific proteins 466 Blue biotechnology 7 Boc-Lys 2-Cl-Z ; -OH 315 Bombesin BB ; 382 Bombilitins 385 Bone Bone-growth-inducing substances 343 Bone marrow stromal cell 113 Bone morphogenic proteins BMP ; 343 Booster PCR 227 BouguerLambertBeer law 520 Bovine kidney virus 475 Bovine papilloma virus 475 Bradford method for protein determination 525 Bradykinin BK ; 364 Brain natriuretic peptide BNP ; 374 Breast feeding prolactin level 397 Brevibacterium ammoniagenes 558 Brevibacterium ammoniagenes 55, 56 Brevibacterium lactofermentum 115 Brewer's yeast 335 Bromoacetyl bromide in enzyme immobilization 553 Bubble aeration in bioreactors 106 Bubble-column in biotechnology 107 Bubble column gassing devices for 674 Buserelin 337 Butadiene from ethanol 821 1-Butanol butyl alcohol ; NBA ; acetone fermentation 820 Butanols production by clostridium spp. on calcium alginate 713 tert-Butoxycarbonyl group Boc ; as amine protecting group in peptide synthesis 314 tert-Butyl dicarbonate 314 tert-Butyl ester group OtBu ; as carboxyl protecting group in peptide synthesis 316 S-tert-Butylthio group StBu ; as sulfhydryl protecting group in peptide synthesis 317 C Cabagin 292 Cadre 294 Calcitonin CT ; 325, 344, 346 Calcitonin gene related peptide CGRP ; Calcitonin L 347 Calcitonin S 347.
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