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1 Cohen, G.M. 1997 ; Biochem. J. 326, 116 2 Budihardjo, I., Oliver, H., Lutter, M., Luo, X. and Wang, X. 1999 ; Annu. Rev. Cell Dev. Biol. 15, 269290 3 Adams, J.M. and Cory, S. 2001 ; Trends Biochem. Sci. 26, 6166 4 Wolter, K.G., Hsu, Y.T., Smith, C.L., Nechushtan, A., Xi, X.G. and Youle, R.J. 1997 ; J. Cell Biol. 139, 12811292 5 Gross, A., Jockel, J., Wei, M.C. and Korsmeyer, S.J. 1998 ; EMBO J. 17, 38783885 6 Gross, A., McDonnell, J.M. and Korsmeyer, S.J. 1999 ; Genes Dev. 13, 18991911 7 Cain, K., Bratton, S.B. and Cohen, G.M. 2002 ; Biochimie 84, 203214 8 Dale, L.B., Bhattacharya, M., Anborgh, P.H., Murdoch, B., Bhatia, M., Nakanishi, S. and Ferguson, S.S. 2000 ; J. Biol. Chem. 275, 3821338220 9 Gu, C., Ma, Y.C., Benjamin, J., Littman, D., Chao, M.V. and Huang, X.Y. 2000 ; J. Biol. Chem. 275, 2072620733 10 Saito, S., Hiroi, Y., Zou, Y., Aikawa, R., Toko, H., Shibasaki, F., Yazaki, Y., Nagai, R. and Komuro, I. 2000 ; J. Biol. Chem. 275, 3452834533 11 Zhu, W.Z., Zheng, M., Koch, W.J., Lefkowitz, R.J., Kobilka, B.K. and Xiao, R.P. 2001 ; Proc. Natl. Acad. Sci. U.S.A. 98, 16071612 12 Chen, C.K., Burns, M.E., Spencer, M., Niemi, G.A., Chen, J., Hurley, J.B., Baylor, D.A. and Simon, M.I. 1999 ; Proc. Natl. Acad. Sci. U.S.A. 96, 37183722 13 Choi, S., Hao, W., Chen, C.K. and Simon, M.I. 2001 ; Proc. Natl. Acad. Sci. U.S.A. 98, 1309613101 14 Koh, J.Y., Palmer, E. and Cotman, C.W. 1991 ; Proc. Natl. Acad. Sci. U.S.A. 88, 94319435 15 Yan, G.M., Lin, S.Z., Irwin, R.P. and Paul, S.M. 1995 ; Mol. Pharmacol. 47, 248257 16 Lindenboim, L., Pinkas-Kramarski, R., Sokolovsky, M. and Stein, R. 1995 ; J. Neurochem. 64, 24912499 17 Leloup, C., Michaelson, D.M., Fisher, A., Hartmann, T., Beyreuther, K. and Stein, R. 2000 ; Cell. Death Differ. 7, 825833, for example, ciprofloxacin.
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Any patients with CFS complain of fever, sore throat, and lymph node swelling and tenderness. Since these symptoms are highly suggestive of microbial disease, many physicians approach patients with CFS, at least in the initial stages, as though infection is a central mechanism or "trigger." It is clear that at least 50% of patients with CFS have an infectious episode as an initial trigger for the syndrome.41 In some cases, the inciting event is clearly a mononucleosis-like illness, occasionally a standard EBV-associated mononucleosis. More often, it is a non-specific upper respiratory infection, a sinusitis or bronchitis, occasionally an influenza-like illness, the latter characterized by severe myalgias, high fevers, and severe malaise. Some patients may actually describe vividly an event at a specific date and time of day when they became ill, relating that after that event they never felt healthy again. Paradoxically, over time for many patients with CFS, the initial symptoms may blur and be difficult to recapture in the medical history obtained years later. Epidemiology Pathophysiology Many infectious agents seem capable of inciting CFS, but few good studies provide an epidemiologic link to specific agents. One early study depicted an outbreak of disease probably associated with human herpes virus 6 HHV-6 ; at Incline Village, Nevada. In this outbreak, there was evidence of depletion of B-cell lymphocytes and development of symptoms consistent with CFS.42, 43 Older observations from Europe suggested that an illness called myalgic encephalomyelitis had occurred in clusters, which suggested an infectious contagious basis for the outbreak.43 Although it is tempting to attribute CFS to an unresolved infection secondary to viral infection, such as mononucleosis or influenza, it is difficult to attribute the constellation of symptoms and signs that currently define CFS as due to a single infectious agent. There is some evidence, however, that CFS may be associated with some unresolved or persistent infectious agents. For example, most patients with CFS have persistently and, at times, markedly elevated antibodies to portions of the EBV, particularly the viral capsid, early antigen, and nuclear antigens. The same can be said, albeit less assuredly, for HHV-6. A certain percentage of patients with highly reactive HHV-6 serology will also have intermittent viremia with HHV-6, the significance of which remains unclear, since there are no studies that have been directed at eradicating HHV-6 from the vascular compartment. Recently, Belgian and French co.
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