Alexeeva LI, Kolesnik TV, Myakotkin BA, Krylov MJu, Demin NV; Institute of Rheumatology RAMS, Moscow, Russia Objective: Osteoarthritis OA ; is a multifactorial disease, so the heritable predisposition plays a key role in the development of this disease. There are some known genetic markers associated with BMD, however their association with OA was detected too. Methods: The study included 179 women residing in Moscow and its region median age 64.1 8.4 ; with OA of knee joints according ACR criteria ; . Control group consisted of 60 women in postmenopausal period median age 68.6 6.6 ; without osteoporosis and OA. BMD was determined by dual-energy X-ray absorptiometry DXA ; with apparatus QDR 4500 Hologic ; at lumbar spine and femoral neck. Radiography of knee joints was done in two planes and assessed by classification of Kellgren-Lawrence. Polymorphism of VDR, ESR, COL2A1 and COLIA2 genes were studied by polymerase chain reaction PCR ; and vertical electropheresis in agarose gel. Results: Based on examination of 179 women with OA the reliable increase of BMD during growth of OA stage in spine I-st 0.848 g cm2, IV-st 0.962 g cm2; p 0.034 ; and femoral neck Ist 0.716 g cm2, IVth- 0.798 g cm2; p 0.025 ; . Reliable differences in distribution of genotypes Fok1 FF ; , Bsm1 BB ; gene VDR, HindIII Hh ; gene COL2A1, Rsal Cc ; gene COLlA2 in OA pts as compared with control OR-2.33, 2.79, 2.52 and 2.62, respectively ; . Carriers of double heterozygotes XxPP, HhCc, PPBB, BBCC, BBHh, PPHh, ppcc have increased risk of OA by 6.5, 6.6, 7.8, which could be regarded as an important risk factor for OA onset. There was no correlation between BMD and the studied genetic markers, with the exception of gene ESR alpha; the association close to significant, r 0.18 ; . BMD in spine and femoral neck ; correlated with osteophytes in the knee joints r 0.4, p 0.05 ; , and size of the osteophytes - with a gene ESR . Conclusion: Obtained data confirm the hypothesis of participation of increased BMD and genes VDR, COL2A1 and COLIA2 in pathogenesis of OA of knee joints.
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Introduction The Sanitarium Health Food Company NZ ; receives between 600 to 800 nutrition enquiries per month. Some are from individuals seeking information on the nutritional importance of eating breakfast. Given this, Sanitarium requested the development of a nutrition based breakfast pamphlet. Objective To develop and produce an accurate and informative final draft, nutrition based breakfast pamphlet for the general New Zealand adult population, explaining the importance of breakfast for good health and ways of increasing breakfast intake in those who currently do not eat breakfast. Methods The first draft was developed, peer reviewed and highlighted changes were incorporated into the second draft. Five focus groups 34 participants ; pretested the second draft. A transcript based analysis, copy-editor analysis and inter-rater assessment was conducted and emerging themes reported. The final draft incorporated changes outlined by the focus group participants with a new standard graphic design concept adopted by Sanitarium. The SMOG Formula was used to test the reading level of the final draft. Results An eye-catching cover, more pictures, larger print and greater contrast between the print and background colours were needed. To ease reading, text laid out in point rather than paragraph form was favoured. The smaller A4 sheet folded into three sections size was preferred and generalised recipes were requested. The message regarding the importance of eating breakfast was well received. Information on how to read nutrition labels, the provision of less expensive breakfast options, an introductory and a summary paragraph were requested. The pamphlet's reading level was acceptable, at a SMOG Score of 11, for the adult consumer. Conclusions Breakfast -- A Smart Way to Start the Day is an informative and practical nutrition based breakfast pamphlet that explains the importance of breakfast for good health and gives practical tips for ensuring a healthy breakfast is regularly eaten. It fills an existing gap in Sanitarium's 'Nutrition Fact Sheet Series' and will be available from Sanitarium's Nutrition Education Service to answer consumer enquiries.
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Difference in weight of white adipose pads between SR141716- and vehicle-treated animals confirmed that weight loss was accompanied by a decrease in adipose tissue. Similar data showing a rapid tolerance to the anorectic action despite a sustained and prolonged effect on body fat loss also being obtained when obese Zucker rats were treated for 14 days with SR141716 59 ; . Importantly, another CB1 receptor antagonist, AM-251, produced similar effects in DIO mice 311 ; . Very recently, Poirier et al. monitored weight and metabolic marker changes in three groups of mice after establishing a condition of obesity by a 5 month high fat diet 309 ; . Two groups of animals were maintained on a high fat diet, but one was treated for 10-weeks with 10 mg kg SR141716 and the other one with a vehicle. A third group received a dietary switch to standard food after the 5 months on high fat diet. SR141716 induced a weight loss of approximately of 78% in comparison to the weight of the animals receiving the vehicle. More importantly, the anti-obesity effect of the drug was equivalent both in terms of time course and maximum effect ; to that achieved by switching obese mice to a normal diet 309 ; . Again, the authors demonstrated that the anorectic effect of the CB1 receptor antagonist vanished with time since the energy intake in the SR141716-treated animals was equivalent to those on a high fat diet during the last 6 weeks of the experiment and significantly greater than in the group receiving standard diet. Consistent with a previous report 310 ; , the SR141716-induced weight loss was accompanied by normalization of leptin, insulin and glucose levels 309 ; . Notably, SR141716 also normalized triglycerides and LDL-cholesterol. Moreover, the HDL-cholesterol LDL-cholesterol ratio following SR141716 treatment was significantly higher than in the other two groups 309 ; . Whether this effect on lipid metabolism is indirectly related to an elevation of adiponectin is still a matter of debate. Sherman et al. recently showed that a 9-day treatment of DIO mice with the CB1 receptor antagonist AM251 increases Uncoupling Protein UCP ; -1 and UCP-3 mRNA expression and meridia.
This document. Data on diversion generally does not include benzodiazepines and may be due to policy decisions, and lack of resources to assess this problem. Toxicology is very expensive and may be limited, even when there is an arrest. Group participants identified an increased need for methodologically sound benzodiazepine information in the following areas: Arrests and seizures Motor vehicle accidents School suspensions Profile of benzodiazepine users The association between benzodiazepines and arrests, drug treatment, falls Adequacy of the ICD-10 Chronic versus acute prescribing Prescription monitoring program to begin July 04 ; o Will include everyone living in Maine who fills a prescription o Will miss illegal sales o Will cover all pharmacies licensed in Maine o Will include only what is in the prescription, not the diagnosis o May or may not have age and gender Why benzodiazepines are being prescribed Benzodiazepine efficacy as well as morbidity, including how much prescribed, how much actually taken, and which particular drugs Prescription guideline monitoring data Trends in abuse Trends in appropriate prescribing among physicians o Focus groups with physicians o What influences a doctor's decision to prescribe o What is inappropriate prescribing especially for elders ; o How to develop index cases of inappropriate prescribing can we use insurance data? ; The discussion then focused on the 2004 survey of benzodiazepines. The following recommendations were made: Do not add newer drugs, but keep the existing benzodiazepine list For prescription data, collect o Age, gender, o Specific drug, dose, and duration o Presenting illness o Characteristics of prescribers and recipients where possible o Respond to other conference break-out groups' needs for data monitoring Generate a statement for Dirigo Health: 1-page about the benzodiazepine problem Continue to participate in the Maine Community Epidemiology Surveillance Network Continue to search for information on counterfeit drugs Work to develop interest in a drug return with the MMA.
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During 2005 and early 2006, Meda acquired several products; for example, Cibacen and Cibadrex from novartis, a Swiss pharma company, for SEK 941 million. Cibacen and Cibadrex are well-established drugs for treating high blood pressure and thus significantly enhance Meda's position in the cardiovascular therapy area. In early 2006, Meda signed another agreement with novartis to acquire the European rights to the Parlodel products. Parlodel is a well-proven specialist drug that sold for about SEK 130 million in 2005. The product is a dopamine agonist and prolactin inhibitor; Meda acquired it as of March 2006. In early 2005, Meda announced its acquisition of Lmovane from sanofi-aventis, a pharma company; Imoavne is an established drug for treating sleeping disorders. by selling its Parallel Trading business area in 2005, Meda streamlined its operations and improved its profitability. The deal resulted in a capital gain of SEK 37 million.
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