A Prescription Drug Program is available to employees and dependents enrolled as plan participants in one of the Arkansas State and Public School Employee Benefits Division EBD ; sponsored medical plans. Prescription drug benefits are not available without participation in one of the medical plans. Retail Prescription Drug Card Program Drugs that are prescribed for short-term use up to a 34-day supply ; should be filled using the retail prescription drug card. The Pharmacy Benefit Manager AdvancePCS ; administers the Retail Prescription Drug Card Program. This benefit is offered in conjunction with one of the Arkansas State and Public School Employee's Medical Plan options. Participants receive a prescription drug card, which may be used to purchase drugs from one of the AdvancePCS network pharmacies. The AdvancePCS network includes over 700 pharmacies in Arkansas and over 20, 000 pharmacies nationwide. Most chain stores such as WalMart, Walgreen's and Kroger participate in this network, as well as many independent pharmacies across the state. Confirmation of participating Pharmacies may be obtained by calling AdvancePCS at 1-877-456-9586 or through its website at : ar.advancerx The medications eligible for coverage will fall into one of three categories: generic, formulary brand, or non-formulary brand. The co-payment amount is dependent upon whether the prescription is for a generic, a formulary brand name preferred ; drug or a non-formulary brand name non-preferred ; drug. A generic drug is identical in chemical composition to its brand name counterpart, has been approved by the Food and Drug Administration FDA ; to be therapeutically equivalent, and is as effective as the brand name product. The use of generics and formulary brand name drugs perform a vital role in controlling the cost of prescription drugs for both the participant and the plan. All non-formulary drugs have alternatives available preferred brand name drugs and possibly generics, both of which are more cost effective. Once a generic medication is released, the brand name counterpart becomes a non-formulary medication. As new medications receive FDA approval and are released, they are reviewed by the AdvancedPCS Pharmacy and Therapeutics committee P&T ; . The P&T Committee makes a recommendation to the Arkansas State and Public School's Drug Utilization and Evaluation Committee DUEC ; . The decision to add or not to add a medication to the formulary is first and foremost based upon efficacy.
81. Wilhelm H, Schaeffel F, Wilhelm B., Age dependence of pupillary near reflex, Klin Monatsbl Augenheilkd. 1993 Aug; 203 2 ; : 110-6. BACKGROUND: This study was performed to gain age correlated normal values for the pupillary near reflex. METHODS: Accommodation and pupillary near reaction were measured by means of simultaneous infrared video retinoscopy and pupillography in 64 healthy volunteers aged between 5 and 55 years. Measurements were done at a reduced, near mesopic, light condition with accommodation to 10, 14, 20 and 33 cm. RESULTS: The pupillary near response varied highly with age: persons younger than 20 years of age showed a significantly smaller pupillary near response as compared to those older than 20 years. In most of the children younger than 10 years the pupil near response was very small less than 10% constriction ; at accommodation distances longer than 10 cm. There was a significant difference between the age groups younger and older than 20 but no statistically significant differences within these age groups. CONCLUSION: A change of the pupillary near reaction takes place around the age of 20. We conclude that this change does not only reflect the aging of the cristalline lens but is due to an age related change of the supranuclear control, for example, ranitidine in pregnancy.
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Antacids are less appropriate for patients with FHB: Their short duration of action means that FHB sufferers would need to take multiple doses per day, which would likely lead to side effects even if patients did adhere to the regimen.11 Another drawback is their inadequacy as heartburn prophylaxis. Histamine H2-Receptor Antagonists--Three types of receptors trigger production of hydrochloric acid in the stomach. H2RAs block one of these receptors--the histamine H2 receptor--on the gastric parietal cell, thereby impeding the formation of hydrochloric acid. H2RAs that are available OTC include cimetidine Tagamet HB 200 ; , ranitidine Zantac 75 ; , famotidine Pepcid AC ; , and nizatidine Axid AR ; . Dosages of these OTC versions are one half of the standard lowest prescription dosage. OTC famotidine was recently approved at the original prescription dosage. ; Although these four agents differ somewhat in potency, they can be used interchangeably according to the ACG.24 They are particularly useful in patients with episodic heartburn who take the medication before an activity that is likely to produce reflux symptoms eg, eating a heavy or spicy meal ; . Comparisons between antacids and the H2RAs are limited, but it has been suggested that the former provide a more rapid response onset of action, 30 minutes vs up to minutes ; , 36 whereas the latter are generally more effective and have a much longer duration of action.24, 36 Efficacy trials have shown that the H2RAs are superior to placebo for the relief of episodic heartburn.37-40 Placebo-controlled studies have also demonstrated the efficacy of these agents in preventing heartburn.41, 42.
Ranitidine hcl GEN FOR ZANTAC ; .10 reclipsen, desogestrel-ethinyl estradiol GEN FOR ORTHO-CEPT ; .12 REESE PINWORM, pyrantel [OTC] .4 repaglinide.10 REQUIP.6 REQUIP, ropinirole hcl [PA] .23 RESCRIPTOR, delavirdine mesylate .4 REYATAZ, atazanavir sulfate Protease Inhibitor submit to State .4 ribavirin [PA] [QLL] GEN FOR REBETOL ; .5 RIDAURA, auranofin .11 rifampin GEN FOR RIFADIN ; .4 rimantadine hcl [QLL] GEN FOR FLUMADINE ; .5 rimexolone.13.
FDA U.S. Food and Drug Administration. Information from references 10 through 29.
WS: Amount mg ; of Eanitidine Hydrochloride Reference Standard Operating conditions-- Detector: An ultraviolet absorption photometer wavelength: 322 nm ; . Column: A stainless steel column 4.6 mm in inside diameter and 20 cm in length, packed with octadecylsilanized silica gel for liquid chromatography 10 mm in particle diameter ; . Column temperature: A constant temperature of about C. 259 Mobile phase: A mixture of methanol and diluted 0.5 mol L ammonium acetate TS 1 in 17: 3 ; . Flow rate: Adjust the ow rate so that the retention time of ranitidine is about 5 minutes. System suitability-- System performance: Dissolve 20 mg of Ranitidinne Hydrochloride and 5 mg of benzalphthalide in 200 mL of the mobile phase. When the procedure is run with 10 mL of this solution under the above operating conditions, benzalphthalide and ranitidine are eluted in this order with the resolution between these peaks being not less than 2.0. System repeatability: When the test is repeated 6 times with 10 mL of the standard solution under the above operating conditions, the relative standard deviation of the peak area of ranitidine is not more than 1.0z and relafen.
Fig. 11. The influence of therapy with L. acidophilus in C.albicans inoculated rats with or with ranitidine on the ulcer healing and GBF. Combined therapy with L. acidophilus in Candida inoculated rats with ranitidine reversed the increase in the ulcer area and accompanying fall in GBF ulcer margin caused by Candida as compared to the respective values determined in Candida inoculated rats without addition of probiotic bacteria.
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Results should be viewed with caution because of the potential for bias regarding blinding of new inhaler devices in clinical trials. Long term parallel trials have shown that regular daily use of hydrofluoroalkane pressurised metered dose inhalers may reduce the requirement for short course oral steroids.48 49 However, this result may be biased because of inadequate randomisation in one trial. Confirmation from further trials is required. Further research Although we did not find significant differences for most outcomes, the confidence intervals could include clinically important differences. Our comparison of population means cannot show such clinically important differences for individual patients from different inhaler devices. Small changes in physiological measures such as pulmonary function will not necessarily be important in themselves, but rather in the impact they have on the symptoms and quality of life of the patient.50 Future trials should address the paucity of patient centred outcomes such as quality of life, adherence, nocturnal awakening, and days off work or school. Further systematic reviews are needed to assess the effectiveness of pressurised metered dose inhalers with or without spacer devices and the effectiveness of training and education about use of inhaler devices. Conclusion We found no evidence that alternative inhaler devices are clinically more effective than pressurised metered dose inhaler for delivery of short acting 2 bronchodilators. Therefore, pressurised metered dose inhalers or the cheapest inhaler device the patient can use adequately should be prescribed as first line in all patients with stable asthma requiring short acting 2 agonist bronchodilators.
00093011201 00093011205 00093011301 CIMETIDINE TAB 300MG CIMETIDINE TAB 300MG CIMETIDINE TAB 400MG CIMETIDINE TAB 400MG CIMETIDINE TAB 800MG CIMETIDINE TAB 800MG CIMETIDINE SOL 300 5ML FAMOTIDINE TAB 20MG FAMOTIDINE TAB 40MG RANITIDINE TAB 150MG RANITIDINE TAB 150MG RANITIDINE TAB 150MG RANITIDINE TAB 150MG RANITIDINE TAB 300MG RANITIDINE TAB 300MG RANITIDINE TAB 300MG TAGAMET TAGAMET TAGAMET TAB 300MG TAB 300MG TAB 400MG 66 3 $769.81 $54.33 $1, 203.31 $299.92 $582.46 $46.90 $310.47 $9, 032.26 $1, 595.55 $14.35 $7, 435.83 $3, 658.35 $1, 951.74 $528.44 $888.55 $1, 606.56 $794.38 $0.00 $1, 947.60 $293.28 $216.99 $0.00 $646.48 $20.50 $254.30 $59.00 $390.45 0.44% 0.02% 0.71% 0.00% 0.11% 0.03% 0.01% 0.00% 0.35% 0.01% 0.17 and risperdal.
Elevated ABR latencies are not caused by developmental retardation Based on interpeak intervals of ABR, the maturation of central auditory processes is completed around the end of the third month in rodents Moore 1985 ; . To confirm that the observed elevated latencies in animals treated with MMI are not a result of retarded maturation of central auditory processes, which may improve during later development, we analyzed ABR latencies in offspring of MMI-treated dams between 3 and 9 mo of age. Auditory brain stem responses were induced by 80 dB peSPL click signals. As shown in Table 4 for animals that underwent a TH-deficient period until P10, there was no further improvement of ABR latencies between 3 and 9 mo of age. This confirms that elevated ABR-interpeak intervals that arise as a result of a short TH-deficient period before the onset of hearing are probably permanent.
Mitoxantrone UF: CL232315 DHAD DHAQ Novantrone NSC-279836 NSC-301739 BT: Anticancer drugs SN: Mitoxantrone is a doxorubicin analog. MK-639 USE Indinavir MK 639 USE Indinavir MKC-422 USE Emivirine MN rgp120 HIV-1 USE rgp120 HIV-1MN Modification of drug therapy EV: Modification du traitement mdicamenteux BT: Drug therapy management Molecular genetic components EV: Composante gntique molculaire BT: Molecular genetics NT: Amino acids Chromosomes Genomes Nucleic acids Peptides Proteins Molecular genetic processes EV: Processus gntique molculaire BT: Molecular genetics NT: Cell division and ritalin.
Failure to respond promptly or fully to such rescue medication, signals a need for urgent medical advice and treatment.
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FIGURE 11. Competition between QX-314 and cocaine. Examples of single-channel records at + 50 the presence of A ; 300 #M cocaine and B ; in the presence of both 300 ttM cocaine and 5 mM QX-314. Both drugs were applied from inside. C ; Distribution of open time with 300 ttM cocaine. The data were measured as described in Fig. 5. N 248, ro 78 ms. D ; Distribution of open time with 300 ttM cocaine and 5 mM QX-314. N 105, ~o 151 ms. Bilayer membrane was formed in a 200-mM symmetrical NaCI solution and serevent.
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| Free RanitidineThe gastrointestinal absorption of cimetidine and ranitidins is accelerated when they are coadministered with cisapride and serzone.
Table 1: The healing rates of duodenal ulcer with ranitidine or antacid therapy of 6 weeks. Ranitidije 150 mg bd Healed Not healed Total 64 80.0 ; % 16 20.0 ; % 80 Antacid 105 ml day 52 77.6 ; % 15 22.4 ; % 67 Test for difference p 0.05 p 0.05.
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Urohypophysis'te Blm Ed.: H. Heller ve B.T. Pickering ; , International Encylopedia of Pharmacology and Therapeutics, 1. Cilt, s. 399, Pergamon, Oxford, 1970. Tommwambingu, H.: Water intoxication and oxytocin. Brit. Med. J. 290: 13, 1985. Tromans, P.M. ve di.: Comparative study of estradiol and prostaglandin E2 vaginal gel for ripening the unfavourable cervix before induction of labour. Brit. Med. J. 282: 679, 1981. X. BLM: OTAKO DLER 86. Histamin ve Antihistaminikler Ahmad, S.R.: Antihistamines alert. Lancet 340: 542, 1992. Arrang, J.M. ve di.: Highly potent and selective ligands for histamine H3 receptors. Nature 327: 117, 1987. Asthon, M.G. ve di.: Healing of gastric ulcers after one, two and three months of ranitidine. Brit Med. J. 284: 467, 1982. Avella, J. ve di.: Effect of histamine H2 receptor antagonist on delayed hypersensitivity. Lancet 1: 624, 1978. Awcuters, F.H.L. ve di.: Pharmacology of the specific histamine H1 antagonist astemizole. Arzneim.Forsch. 33. 381, 1983. Axelrod, J. ve F. Hirata: Phospholipid methylation and receptor induced release of histamine from cells. TIPS 3: 156, 1982. Bardhan, K.D. ve di.: Does treatment with cimetidine extended beyond initial healing of duodenal ulcer reduce the subsequent relapse rate. Brit. Med. J. 284: 621, 1982. Bays, D.E. ve R. Sables: Recent developments in the use of H2 antagonist to treat peptic ulcers. Pharmacy Int. 4: 199, 1983 Beaven, M.A.: Histamine. N. Engl. J. Med. 294: 30 ve 320, 1976. Block, J.W. ve di.: Definition and antagonism of histamine H2 receptors. Nature 236: 385, 1972. Brogden, R.N. ve di.: Cimetidine a review of its pharmacological properties and therapeutic efficacy in peptic ulcer disease. Drugs 15: 93, 1978. Cargill, J.M. ve di.: Very longterm treatment of peptic ulcer with cimetidine. Lancet 2: 1113, 1978. Chand, N. Distribution and classification of airway histamine receptors: The physiological significance of histamine H2 receptors: Adv. Pharmacol. Chemother. 17: 103, 1980. Cheng, H.C. ve J.K. Woodward: Antihistamine effect of terfenadine. Drug. Dev. Res 2: 181, 1982. Clain, J.E.: Diagnosis and management of gastrinoma ZollingerEllison syndrome ; . Mayo Clin. Proc. 57: 265, 1982. Cohen, A.F. ve di.: The acute effects of acrivastine BW 825 C ; , a new antihistamine, compared with triprolidine on measures of central nervous system performance and subjective effects. Clin. Pharmacol Ther. 38. 381, 1985. Czerwonka, R. ve di.: Release of histamine by H2 receptor antagonists. Lancet 2: 216, 1987. Danilewitz, M. ve di.: Ranitidie supression of gastric hypersecretion resistant to cimetidine. N. Engl. J. Med. 306: 20, 1982. DeFeudis, F.V.: Studies on endotheliumdependent vasorelaxation. Gen. Pharmacol 17: 1, 1986. Dale, M.M. ve J.C. Foreman: Textbook of Immunopharmacology, 2nd Ed., Blackwell, Oxford, 1989. Dial, E. ve di.: Isolated parietal cells: histamine response and pharmacology. JPET 219: 585, 1981. Donoso, A.O. ve M. Barontini: Increase in plasma catecholami and singulair.
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Correspondence to: Dr. Deenan Pillay Public Health Laboratory Service Antiviral Susceptibility Reference Unit Department of Infection Medical School University of Birmingham Edgbaston Birmingham B15 2TT Tel Fax: 0121 414 6957.
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