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In a double-blind, double-dummy dose reduction study, 197 adult patients with asthma stabilised during treatment with bdp 2000 µ g day were randomised to receive pulmicort ® turbuhaler ® or fluticasone diskus™ accuhaler™ 800 µ g day and the dose was halved at 5-week intervals if asthma control was maintained kuna et al, 2003.
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Hospital-based physicians, hospitals, emergency rooms and free-standing surgical centers in the United States. Specialty Products Hokunalin Patch is the world's first and only tape formulation for the management of asthma, and posted the highest sales of BASF Pharma's specialty products for niche markets. Currently sold in Japan only, the Hokunalin Patch contains the active ingredient tulobuterol which is transmitted from the patch through the patient's skin. Due to the product's successful performance in Japan, BASF Pharma believed that the Hokunalin Patch would have a strong potential for being registered and marketed internationally. Pharmaceutical Active Ingredients As part of BASF Pharma's new orientation in 2000 to focus on the research, development and production as well as the marketing and sales of prescription medications, the pharmaceutical active ingredients business was transferred to BASF's Fine Chemicals division on July 1, 2000. Generic Pharmaceuticals As part of BASF Pharma's new business focus in 2000, the company sold its generic pharmaceuticals business to the generics division of Novartis, effective January 1, 2001. BASF Pharma's generics business was focused on European markets. Sales The Pharmaceuticals division's sales to third parties were 02, 526 million in 2000. Ethical drugs, which BASF defines as drugs that require a prescription from a physician and which are sold under an individual brand name, represented BASF Pharma's core business in 2000. Sales of these drugs increased 16% in 2000 to 02, 485 million. The following table shows BASF Pharma's ethical drugs sales for 2000 by therapeutic field.
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ABSTRACT OBJECTIVE: To compare asthma-related health care resource utilization among a matched cohort of asthma patients using inhaled corticosteroids ICSs ; plus either montelukast MON ; or salmeterol SAL ; as combination therapy for asthma, during a time prior to the availability of fixed-dose combinations of ICS SAL. METHODS: A retrospective analysis using the PHARMetrics patient-centric claims database was conducted for the period preceding the market introduction of combination fluticasone-SAL in September 2000. Patients had to meet the following criteria for inclusion in the study: they had to be between the ages of 4 and 55 years; they had to have been continuously enrolled for 2 years; they had to have initiated ICS MON or ICS SAL therapy between July 1, 1998, and June 30, 1999; and they had to have had either a ; a diagnosis of asthma based on International Classification of Diseases, Ninth Revision, Clinical Modification ICD-9-CM ; codes of 493.xx ; for 2 outpatient visits, 1 or more emergency department ED ; visits, or 1 or more hospitalizations within 1 year or b ; pharmacy claim records that contained a National Drug Code for an antiasthma medication betaagonist, theophylline, ICS, cromolyn, or leukotriene ; 2 or more times within 1 year. ICS MON and ICS SAL patients were matched 1 to 1 age and propensity score. Outcomes included asthma-related hopitalizations and ED visits with ICD-9-CM codes of 493.xx, and oral corticosteroid OCS ; fills and short-acting beta-agonist SABA ; fills. Multivariate regression analyses were performed. Subgroup analyses based on sequential or concurrent initiation of combination therapy were also conducted. RESULTS: A total of 1, 216 patients were matched ICS MON 608; ICS SAL 608 ; . Decreased odds of ED visits and or hospitalizations were observed with ICS MON adjusted odds ratio [OR] 0.58; 95% confidence interval [CI], 0.350.98 ; versus ICS SAL. The odds of postindex OCS fills were not different for ICS MON and ICS SAL patients adjusted OR 1.04; 95% CI, 0.79-1.38 ; . Postindex pharmacy claims for SABAs were significantly higher among ICS MON patients versus ICS SAL patients adjusted relative risk [RR] 1.33; 95% CI, 1.17-1.52 ; , and this difference remained regardless of prior use or no prior use of ICSs. In subgroup analyses, mean change in SABA fills varied by how combination therapy was initiated, with sequential addition of asthma controllers leading to a reduction in SABA fills in both groups. For patients with concurrent initiation of combination therapy, the odds of ED visits hospitalizations were significantly lower in patients initiating ICS MON adjusted OR 0.25; 95% CI, 0.08-0.79 ; . CONCLUSION: In this matched cohort, use of ICS MON compared with ICS SAL resulted in similar odds of OCS fills, decreased odds of ED visits and asthmarelated hospitalizations, but higher utilization of SABA. KEYWORDS: Asthma, Montelukast, Salmeterol, Inhaled corticosteroids, Combination therapy, Propensity scoring, Leukotriene and theophylline.
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When comparing treatments with pooled baseline, overnight urinary cortisol creatinine was significantly depressed by both the high and medium doses of fluticasone propionate and mometasone furoate: geometric mean fold suppression 95% CI for difference ; from baseline: fluticasone propionate 2000 g : 1.85 1.21 2.82 ; , p 0.002; fluticasone propionate 1000 g : 1.45 1.07 1.96 ; , p 0.02; mometasone furoate 1600 g: 1.92 1.26 2.93 ; , p 0.001; mometasone furoate 800 g 1.39 1.04 1.88 ; , p 0.02 Individual patient data shown in Figure 2 ; . Values for the low doses of ICS were as follows: fluticasone propionate 500 g: 1.06 0.80 1.41 ; , p 1.00; mometasone furoate 400 g: 1.17 0.88 1.55 ; , p 0.54. There was a similar pattern for uncorrected overnight urinary cortisol Figure 3 ; , with data shown in table 3 and spironolactone.
All potential angioedema events, including any swelling of the head or neck, were reported as potential study end points. Detailed follow-up information regarding the intensity, clinical features, severity, treatment, outcome, etiology of the event, as well as a description of diagnostic procedures, prodromal symptoms, concomitant medications, and a history of compliance with the study drug were obtained using a structured questionnaire. All data were then reviewed in blinded fashion by the Event Adjudication Committee a panel of experts in angioedema ; to determine whether angioedema had occurred. Three primary adjudicators reviewed each event and met to reach a consensus on the presence or absence of angioedema, the severity class, and the etiology of the event. Agreement by 2 of primary adjudicators was necessary and sufficient to make a determination. Because no standardized or validated classification system for the severity of angioedema is currently available, a classification system was developed for this purpose. This system used treatment variables as the primary basis for classification, in the belief that the treatment provided would reflect the severity and clinical importance of the signs and symptoms of angioedema. Thus, the Event Adjudication Committee classified the severity of angioedema according to an ad hoc scale that used measures of treatment intensity as proxies for severity. Angioedema events were classified by severity as class 1 when patients were given no treatment or were given antihistamines only, class 2 when patients were treated with catecholamines or corticosteroids, class 3 when patients were hospitalized but no mechanical airway protection was given subclassified to class 3a if patients were hospitalized with no airway compromise or class 3b if they were hospitalized with airway compromise ; , and class 4 when patients required airway protection or died, because fluticasone 50 mcg.
27. "Can We Predict Outcomes?" Issues and Controversies in Prostate Care, Ontario Regional Meeting, Scottsdale, Arizona, May 19, 2001. 28. "Statistical Models Predicting Relapse After Local Treatments." 1st International Prostate Cancer Conference, San Juan, Puerto Rico, June 27, 2001. 29. "Decision Making Tables, Which Are Best, And How Accurate Are They?" British Columbia Regional International Conference on Prostate Cancer Meeting, Quadra Island, B.C., Canada, August 18, 2001. 30. "Prostate Cancer Nomograms: A Tool for Decision Making." US TOO! Prostate Cancer Survivors Support Group, Memorial Sloan-Kettering Cancer Center, New York, October 18, 2001. 31. "Nomograms for Predicting Outcomes." Informatics Solutions for Prostate Cancer, Toronto, October 27, 2001. 32. "Nomograms in the Management of Prostate Cancer." Thomas Jefferson University 12th Annual Urology Update Symposium, Philadelphia, November 1, 2001. 33. "Prostate Cancer Nomograms: A Tool for Decision Making." MAN to MAN Prostate Cancer Survivors Support Group, American Cancer Society, New York, November 13, 2001 34. "Predicting Clinical Outcome in Prostate Cancer: Nomograms and Other Strategies." Department of Urology Grand Rounds, Columbia University, New York, January 31, 2002. 35. "How To Know When You Should Use A Prognostic Model." CaPSURE 2002 Investigators Meeting, San Diego, California, March 9, 2002. 36. "Predictors of Disease Progression in Prostate Cancer." 1st Annual Opinion Leader Summit, Barcelona, April 18, 2002. 37. "Approaches Toward Outcomes Analysis." 23rd American Brachytherapy Society Meeting, Orlando Florida, May 24, 2002. 38. "Algorithms for Prostate Cancer: From Diagnosis to Outcomes." 2nd International Prostate Cancer Congress, St. Thomas, U.S.V.I., July 19, 2002. 39. "Predictive Models of Prostate Cancer Progression." Prostate 2002 International Conference, Molecular Mechanisms as Targets in Prostatic Diseases, Liverpool, England, September 19, 2002. 40. "Prostate Cancer Nomograms." Dana-Farber Cancer Institute's Genitourinary Oncology Seminar Series, Boston, Massachusetts, October 2, 2002. 41. "Nomograms and Prediction Models in Prostate and Other Cancers." Population Science Conference, Fox Chase Cancer Center, Philadelphia, November 12, 2002. 42. "Predicting Outcomes: Innovative Use of Neural Networks and Nomograms." Innovations and Challenges in Prostate Cancer: Prevention, Detection and Treatment, Cambridge, Massachusetts, November 15, 2002. 43. "Nomograms as Counseling Tools." Society for Urologic Oncology Annual Meeting, Bethesda, Maryland, December 13, 2002. 44. "Should We Predict Health Outcomes The Way We Predict Financial Outcomes?" American Statistical Association, New York, New York, December 17, 2002. 45. "Nomograms for Cancer Prediction" MD Anderson, Department of Biostatistics, Houston, Texas, February 5, 2003. 46. "Modeling Patient Prognosis." MD Anderson 10th Annual Genitourinary Oncology Conference, Houston, Texas, February 6, 2003. 47. "Risk Assessment in Prostate Cancer." NCCN 8th Annual Conference. Hollywood, Florida, Friday, March 15, 2003. 48. "The Predictive Value of Nomograms Used in Conjunction with Surgery Hormonal Therapy." Takeda European Medical Congress for Urology. Budapest, Hungary, March 29, 2003. 49. "The Predictive Value of Nomograms Used in Conjunction with Brachytherapy and Radiotherapy." Takeda European Medical Congress for Urology. Budapest, Hungary, March 30, 2003. 50. "Predicting Risk with the Use of Nomograms." Charles Huggins Symposium. Chicago, IL, April 25, 2003 and glimepiride.
For more detailed information about your Horizon Medicare Rx Plan 1 prescription drug coverage, please review your EOC and other plan materials. If you have questions about Horizon Medicare Rx Plan 1, please call Customer Service at 1-866-236-7376, 24 hours a day, 7 days a week. TTY TDD users should call 1-866-2361069. Or, visit HorizonBlue medicare . If you have general questions about Medicare prescription drug coverage, please call Medicare at 1-800-MEDICARE 1-800-633-4227 ; 24 hours a day 7 days a week. TTY TDD users should call 1-877-486-2048. Or, visit medicare.gov, for example, fljticasone proprionate nasal spray.
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Occupational hazards: any psychoactive drug may impair judgment, thinking, or motor skills, and patients should be advised to avoid driving a car or operating hazardous machinery until they are reasonably certain that the drug treatment does not affect them adversely.
Gov site 1 2 3 next » view more » latest news latest news advair hfa ffluticasone propionate and salmeterol xinafoate ; aerosol, metered dailymed drug label updates for the last seven days since - 5 days ago view 13 more » trusted sources trusted sources tapering fluticasons salmeterol dosage after inducing asthma control background - a number of studies have shown that persistent asthma pa ; can often be controlled by combination therapy with an inhaled and panadol.
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Given the similar basic mechanisms of action of beta2-agonists, it is possible that the findings seen in this study may be consistent with a class effect. * Estimated by adding .5 to each cell of the treatment by event occurrence table. Clinical Studies in COPD A long term 52 week ; clinical study in 1465 COPD patients evaluated the safety and efficacy of ADVAIR DISKUS 50 500 salmeterol xinafoate fluticasone propionate ; versus placebo and the individual components salmeterol 50 mcg and fluticasone 500 mcg ; , all taken twice daily via the DISKUS inhalation device. Patients who had an established clinical history of COPD with a pre-bronchodilator FEV1 of 25 to 70% of predicted normal, poor reversibility of airflow obstruction defined as an increase of 10% of the predicted normal FEV1 value following the administration of 400 mcg salbutamol ; , and pre-bronchodilator FEV1 FVC ratio of 70% were included in the study. Patients who had respiratory disorders other than COPD, those requiring long term oxygen or those who received inhaled or systemic corticosteroids or antibiotic therapy in the 4 weeks prior to study start were excluded. The primary measure of efficacy was pre-bronchodilator FEV1. Pre-bronchodilator FEV1 in the ADVAIR DISKUS 50 500 group was 133mL higher than the placebo group p 0.001 ; , 73mL higher than the salmeterol 50 mcg group p 0.001 ; and 95 mL higher than the fluticasone 500 mcg group p 0.001 ; throughout the treatment period. Disease-specific quality of life was assessed with the St. George's Respiratory Questionnaire SGRQ ; . With ADVAIR DISKUS 50 500, the raw mean changes in Total Score ranged from -2.4 at Week 2 to -4.5 at Week 52. A clinically meaningful change of 4.0 was achieved as early as 8 weeks with ADVAIR DISKUS 50 500 but not with placebo, salmeterol 50 mcg or fluticasone 500 mcg. The overall incidence of adverse events and COPD-related adverse events was similar across the four groups during the treatment period. Most commonly reported drugrelated adverse event was candidiasis of the mouth and throat ADVAIR DISKUS 50 500 mcg, 6%; fluticasone 500 mcg, 6%; salmeterol 50 mcg, 1%; placebo, 1% ; . Lower respiratory tract infections and pneumonia occurred in 7% of patients in the placebo and salmeterol groups compared to 12% and 14% in the fluticasone propionate 500 mcg and ADVAIR DISKUS 50 500 mcg groups respectively. No clinically significant effects were observed following any treatment on ECG findings, vital signs or bruise count. Bone density and fracture rates were not assessed in this study and anafranil.
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