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In 2004, the Danish Medicines Agency launched another project, in cooperation with the Danish Pharmaceutical Association and with financial support from Apotekerfonden, to expand the database by incorporating herbal medicines. `The aim is to enable the database to handle interactions between herbal and traditional medicinal products. The project is still ongoing, and we expect to complete it in the course of 2005, ' says Bjarke Naver, human biologist and interaction database assistant.
Aceon Aciphex QL QD Activella Actonel QL Actonel with Calcium QL Actoplus Met QL Actos QL Adderall XR QL Adoxa Dosepack Tier 3 ; Advicor Aldara Alesse Alphagan P QL Altace Altoprev QL QD Androderm Androgel Antabuse Antara Aricept QL Aricept ODT QL Arimidex Arixtra QL Asacol Astelin QL Atrovent Inhaler Avandamet QL Avandaryl QL Avandia QL Avonex QL Azelex Bactroban Cream, Nasal Ointment Benicar QL QD Benicar HCT QL QD Benzamycin Betaseron QL Betoptic S Biaxin XL BiDil Boniva QL Butorphanol Nasal Spray QL Cabergoline Canasa Capex Shampoo Carac Cream Cardizem LA Cefprozil Cellcept Cenestin Ciprodex Clarithromycin Suspension Cleocin Vaginal Suppositories Climara QL Clindesse Colazal Colestid Tablets Copaxone QL Coreg Cortef 5, 10mg Coumadin Cozaar QL QD Crestor QL QD Dapsone Depakote Depakote ER Depakote Sprinkle Differin N Dilantin Diltiazem Sustained Action Capsule Diltiazem Sustained Release 24 Hour Capsule Diovan QL QD Diovan HCT QL QD Dovonex Effexor XR QL Efudex Cream Elestat Enablex QL Entocort EC Esclim QL Estraderm QL Estratest Estratest H.S. Estring QL Evista Femara Fentanyl Citrate Lollipop QL QD, N Fentanyl Transdermal System QL QD Fexofenadine QL QD Fortical QL Fosamax QL Fosamax Plus D QL Fosinopril with Hydrochlorothiazide Fosrenol Gabitril Geodon Glipizide with Metformin Glucagon Emergency Kit Glyburide with Metformin Glycopyrrolate Grifulvin V Tablet Humatrope QD, N Hyzaar QL QD Intal QL Isotretinoin Keppra Ketek Kytril QL, N Lamisil Tablet QL, N Lanoxin Lantus Vials Leuprolide Levaquin Lidoderm Lindane Lipitor QL QD Lofibra Tablet Lovenox QL Lumigan QL Malarone Mesalamine Enema Methergine Metrogel Metrolotion Metronidazole Vaginal Gel Micardis QL QD Micardis HCT QL QD Minocycline Mirapex Moexipril Nabumetone Nasonex QL Neoral Neupogen Niaspan Norditropin QD, N Norvasc Novolin Pens Cartridges Novolog Pens Cartridges Nutropin QD, N Nuvaring Omeprazole QL QD Omnicef QL Ondansetron QL, N Optivar Orphenadrine Orphenadrine Compound Ortho-Prefest Oxandrolone Oxycontin QL QD Oxytrol Paroxetine QL Pegasys QL, N Peg-Intron QL, N Plavix Prandin QL Pravasttain QL QD Precose Premarin Premphase Prempro Prevacid Solutab QL QD Prevpac QL Procrit QD Proctofoam-HC Prograf Prometrium.
Drug Analysis Commentary Commentary Commentary Impression Anthropology Commentary Feature Feature Feature Feature Commentary Feature Feature Feature Andera K Andera K Anderson D Anderson D Anon Anon Anon Anon Anon Anon Anon Anon Anon Anon Anon Microchemical Identification of Gamma Hydroxybutyrate GHB Letter: Microcrystals Used to Identify GHB Letter: Graphics Missing Letter: Straight Up! Fingerprint Enhancement Forensic Anthropology Weasel Words 134 Say Farewell to Jerry Chisum Steve Ojena Retires After 25 Years Fall 1998 Seminar Abstracts Spring 1999 Seminar Abstracts What is a Criminalist? Spring 2001 Seminar Abstracts Fall 2001 Seminar Abstracts Training and Resource Committee 2002-2003 Member Survey Results 2nd Q 2002 4th Q 1999 3rd Q 2000 Summer 1996 2nd Q 2003 Spring 1996 Summer 1996 Winter 1997 1st Q 1999 1st Q 1999 2nd Q 1999 3rd Q 1999 2nd Q 2000 3rd Q 2001 1st Q 2002.
Expression by 26% and did not diminish its 6-fold increase by 22 R ; -OHC. On the other hand, pravastatin did not affect mRNA levels of Abca5, another member of ABCA subclass data not shown ; . Because.
Polyethylene glycol 3350 powder generic Miralax ; .22 polyethylene glycol electrolytes Golytely ; .22 polyethylene glycol electrolytes solution generic Colyte, Trilyte ; .22 polymyxin trimethoprim .12 Ponstel see mefanamic acid Portia .10 posaconazole Noxafil ; .14 potassium chloride .9 potassium chloride K-Dur ; .9 potassium chloride Micro-K, Klor-Con ; .9 potassium iodide SSKI ; .11 potassium phosphate sodium phosphate K-Phos ; .9 Prandin .8 pravastatin .9 Pravachol see pravastatin pravastatin aspirin Pravigard ; .9 Pravigard.9 praziquantel Biltricide ; .14 prazosin .7, 22 prazosin polythiazide Minizide ; .7 Precose .8 prednicarbate .21 prednisolone .12, 15 prednisolone Inflamase Mild ; .12 prednisolone sulfacetamide .12 prednisolone sulfacetamide Blephamide ; .12 prednisone .15 Prefest .11 pregabalin Lyrica ; .18 Premarin cream.11 Premphase .11 Prempro .11 PremproLo .11 prenatal multivitamin .9 prenatal multivitamin NataChew, Natafort, Vitafol + DHA ; .9 Prevacid .18, 21 Prevacid Naprapac .18, 21 Previfem .10 PrevPac.21 Priftin .15 Prilosec see omeprazole Prilosec OTC .21 primaquine .14 primidone .18 primidone Mysoline ; .18 ProAir HFA.23 ProAmatine see midodrine probenecid .15 procainamide.7 procainamide Pronestyl, Procanbid ; .7 Procanbid .7 procarbazine Matulane ; .15.
340459 form a linker to the soluble C-terminal catalytic region residues 460888 ; [4]. A recombinant catalytic fragment, HMG-CoAR 426888 ; , crystallizes as a tetramer, which is effectively a dimer of dimers, with the four active sites located at monomer interfaces. The binding site for HMG-CoA is predominantly on one monomer, which is adjacent to the NADPH-binding site on another monomer. A recent publication describes the three-dimensional structures of human HMG-CoAR in complexes with six different statins [5]. The enzyme-catalysed reaction Figure 1 ; probably involves His-866 and Glu-559 both donating protons [4]. The role of Glu-559 appears to be facilitated by the proximity of Asp-767, which may elevate the pK a of the carboxylate so that a higher proportion is protonated at physiological pH. Several HMG-CoAR inhibitors have been developed as cholesterol-lowering therapeutic agents. Known as statins, they are administered either as salts of carboxylic acids or as lactones, which undergo a ring-opening reaction to generate the inhibitory acids in vivo [6]. In part, the inhibitory acids have structural similarity to the tightly bound catalytic intermediate, mevaldyl-CoA Figure 1 ; . Rosuvastatin CRESTOR R ; is a new statin, originally identified and developed by Shionogi and Company. In assays that measure cholesterol biosynthesis, it is a more potent inhibitor than several other statins in rat hepatocytes IC50 0.2 nM, compared with 1.26.9 nM for atorvastatin, simvastatin, cerivastatin, fluvastatin and pravastatin ; [7]. Rosuvastatin is taken up selectively into the liver after intravenous administration to the rat, where it has a prolonged and prograf.
CI, 0.44-1.27 ; . Risk estimates for pravastatin use 7 cases and 16 controls ; showed no significant difference from the risk estimates for all statins OR, 0.35; 95% CI, 0.14-0.86 ; . When educational level as a marker of socioeconomic status ; was added to the model, the risk estimates for statin use remained unchanged OR, 0.62; 95% CI, 0.46-0.85 ; . Neither adjustment for smoking status OR, 0.60; 95% CI, 0.44-0.82 ; nor adjustment for nonsteroidal anti-inflammatory drug use OR, 0.65; 95% CI, 0.48-0.89 ; changed the risk estimates for statin use. When control group 1 was included in the analysis, statin use was also associated with a decreased risk of lymphoma OR, 0.55; 95% CI, 0.41-0.73 ; . Figure 1 shows the risk of lymphoma among users of statins by type of study and country, using population-based and non statin-related controls group 2 ; only. Statin use was inversely associated with risk of lymphoma both among population and hospital-based studies OR, 0.53; 95% CI, 0.33-0.85 and OR, 0.72; 95% CI, 0.48-1.09, respectively ; . There was heterogeneity among countries for the effect of statins on lymphoma risk P heterogeneity 0.03 ; . The heterogeneity was not due to a different pattern between centers but rather to a different magnitude of the inverse association. A sensitivity analysis, excluding countries one by one, provided consistent.
Health news health videos opinions forum contact fda approves first generic pravastatin, usa main category: statins news article date: 27 apr 2006 - 0: 00 pdt email to a friend printer friendly view write opinions rate article newsletters visitor ratings: healthcare professional: general public: rate this article the food and drug administration today approved the first generic version of bristol-myers squibb's pravachol pravastatin sodium tablets ; , an important step in the agency's effort to increase the availability of lower-cost generic medications and tacrolimus.
Fig. 7. Northern blotting and quantitative analysis of transforming growth factor TGF ; - 1 mRNA expression. TGF- 1 mRNA expression was significantly increased in rat kidneys with CsA treatment at 1 or wk, whereas pravastatin markedly decreased its expression in a dosedependent manner. Relative TGF- 1 mRNA values are represented with the VH group designated as 100% reference and are normalized to 18S. #P 0.01 vs. VH. , P 0.01 vs. CsA. * P 0.05 vs. CsA P5.
This is an alternative technique for establishing iv access in pediatric patients in whom peripheral iv access is difficult and time consuming and pantoprazole.
Is to some extent compensated for by increased urinary excretion. In any event, the contribution of CLR to CL was small even in the TR- animals about 8% ; , indicating that other transporters are capable of mediating the biliary excretion of pravastatin in the absence of Mrp2.
25 relation of diet to cardiovascular disease risk factors in subjects with cardiovascular disease in australia and new zealand: analysis of the long-term intervention with pravastatin in ischaemic disease trial and pentoxifylline.
Drug: bay 12, 9566 metaloprotease inhibitor ; 1999- 2002 sub-investigator sponsor: parke davis pfizer protocol: beyond endorsed lipid lowering with ebct scanning acronym: belles drug: atorvastatin and pravastatin 1999- 2001 sub-investigator protocol: a 20-week open-label assessment of the safety and efficacy profile of atorvastatin when used to optimally control dyslipidemia in postmenopausal patents.
Table 4.37: Novartis' dermatology product portfolio, 2004-5 and trental.
Salazosulfapyridine, balsalazide, cancer risk, colorectal carcinoma, enteritis, mesalazine, olsalazine, 608 sarcoma, chromosome translocation, oncoprotein, transcription factor, 493 scalp, brain metastasis, squamous cell carcinoma, 476 second cancer, adenosquamous carcinoma, parotid gland carcinoma, 534 - antineoplastic agent, cancer adjuvant therapy, Ewing sarcoma, 494 - breast carcinoma, breast surgery, 699 sedation, bone marrow biopsy, childhood cancer, general anesthesia, lumbar puncture, 445 selective estrogen receptor modulator, estrogen receptor beta, heterocyclic compound, structure activity relation, 402 semen analysis, cancer incidence, male infertility, testis cancer, 671 sentinel lymph node, axillary lymph node, breast carcinoma, lymph node metastasis, 698 - foot, lower leg, melanoma, 748 sentinel lymph node biopsy, melanoma, 480 serosa, carcinoma, estrogen receptor, immunohistochemistry, malignant mesothelioma, peritoneum mesothelioma, progesterone receptor, 615 serotonin 3 antagonist, antiemetic agent, antineoplastic agent, aprepitant, chemotherapy induced emesis, neurokinin 1 receptor antagonist, prophylaxis, 435 sertraline, breast cancer, fluoxetine, paroxetine, 724 sexual orientation, breast cancer, social support, 710 short hairpin RNA, cisplatin, drug sensitization, small interfering RNA, uterine cervix cancer, virus infection, 677 signal transduction, antigen recognition, antigen specificity, CD40 ligand, chronic lymphatic leukemia, T lymphocyte, 569 - growth factor receptor, protein, transcription regulation, 416 simvastatin, antilipemic agent, colorectal carcinoma, hydroxymethylglutaryl coenzyme A reductase inhibitor, mevinolin, pravastatin, 604 single nucleotide polymorphism, BRCA2 protein, breast carcinoma, cancer genetics, Rad51 protein, 728 sinus venosus, magnetic resonance angiography, meningioma, neurosurgery, phlebography, vascular patency, 461 sirtuin, cancer inhibition, DNA damage, protein p53, transcription regulation, tumor suppressor gene, tumor suppressor protein, 417 skin cancer, gelatinase A, melanoma, stroma cell, 488 - skin conductance, spectrometry, 492 skin carcinoma, CD8 antigen, cytotoxic T lymphocyte, protein Section 16 vol 143.2.
B. Awareness of ACC's advocacy efforts. Members generally have limited awareness of the full scope of ACC advocacy efforts. They do not blame the College for the problems with health care delivery and reimbursement, but they do want the College to do much more to advocate for change. C. Importance of ACC chapters. Members believe strengthening the state chapters would address both the needs for more effective advocacy and for bridging the leadership-membership gap. It was suggested that the local governors solicit members on a regular basis to gather feedback about key concerns. D. Importance of state local advocacy. Members believe it is at the state and local level that ACC can have the greatest impact in terms of fighting adverse legislation, passing important regulations, negotiating with thirdparty payers, etc and pheniramine.
Yes, it' s safer than statins - popular ones are lipitor, zocor, pravachol, lescol, advicor, crestor and mevaco - newsday warning over grapefruit juice and cholesterol pills may 10, 2006 the grapefruit hazard is not significant for other statins, such as novartis ag' s lescol, bristol-myers squibb co' s pravachol and astrazeneca plc' s cresto - daily mail - uk statin alternatives may 8, 2006 currently, six statins are approved by the fda: atorvastatin lipitor ; , fluvastatin lescol ; , lovastatin mevacor ; , pravasgatin pravachol.
Of cardiovascular events and death with parvastatin in patients with coronary heart disease and a broad range of initial cholesterol levels. N Engl J Med. 1998; 339: 1349-1357. Robins SJ, Collins D, Wittes JT, et al. Relation of gemfibrozil treatment and lipid levels with major coronary events: the VA-HIT: a randomized controlled trial. JAMA. 2001; 285: 1585-1591. Sacks FM, Tonkin AM, Shepherd J, et al, for the Prospective Pravastatni Pooling Project Investigators Group. Effect of pravxstatin on coronary disease events in subgroups defined by coronary risk factors. Circulation. 2000; 102: 1893-1900. Haffner SM, Alexander CM, Cook TJ, et al, for the Scandinavian Simvastatin Survival Study Group. Reduced coronary events in simvastatin-treated patients with coronary heart disease and diabetes or impaired fasting glucose levels. Arch Intern Med. 1999; 159: 26612667. Despres JP, Lamarche B, Mauriege P, et al. Hyperinsulinemia as an independent risk factor for ischemic heart disease. N Engl J Med. 1996; 334: 952-957. Lempiainen P, Mykkanen L, Pyorala K, et al. Insulin resistance syndrome predicts coronary heart disease events in elderly nondiabetic men. Circulation. 1999; 100: 123-128. Meigs JB, Nathan DM, Wilson PWF, et al. Metabolic risk factors worsen continuously across the spectrum of nondiabetic glucose tolerance: the Framingham Offspring Study. Ann Intern Med. 1998; 128: 524533. Robins SJ, Collins D, Rubins HB. Diabetes, hyperinsulinemia, and recurrent coronary events in the VAHigh-Density Lipoprotein Intervention Trial VAHIT ; . Circulation. 2000; 102: II-847. 18. Kuczmarski RJ, Flegal KM, Campbell SM, Johnson CL. Increasing prevalence of overweight among US adults: the National Health and Nutrition Examination Surveys, 1960 to 1991. JAMA. 1994; 272: 205-211. Seidell JC. The worldwide epidemic of obesity: 8th International Congress on Obesity. In: Guy-Grand B, Ailhaud G, eds. Progress in Obesity Research. 8th ed. London: John Libbey; 1999: 661-668. 20. Amos AF, McCarty DJ, Zimmet P. The rising global burden of diabetes and its complications: estimates and projections to the year 2010. Diabetic Med. 1997; 14 suppl ; : S1-S85 and progesterone.
Pravastatin fermentation
Table 2 presents comparative pharmacokinetics of these agents.
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At the Cutting Edge Conference this year, I gave a presentation about research that I have begun looking into spirituality and alcohol and drug treatment. With a cross-section of alcohol and drug workers in the audience, I took the opportunity to take an informal survey of the attitudes and beliefs of New Zealand A & D workers. I was intrigued by American studies that suggested that mental health professionals may be less religious than the normal population, and less spirituallyinclined than their patients Goldfarb, et al., 1996; Weaver, et al., 1998; Galanter, 1999 ; . These studies were limited to medical students, psychologists and psychiatrists respectively. Recent surveys in New Zealand have indicated that 60% of the population believe in God and a further 20% believe in some sort of higher power. One-fifth of New Zealanders pray daily and attend religious services more than once a month, while nearly a third never attend services of worship. While we are less religious than the United States where about 90% of its citizens believe in God, we are similar in religiosity to the United Kingdom Greeley, 1993 ; . At the conference I presented data collected from the first 39 participants in my project. They showed similar patterns of belief in God, daily prayer and attendance of religious services to the normal population, but there was a slightly larger proportion believing in a higher power and noticeably more who never attended services of worship. Ninety surveys were handed out at Cutting Edge and 58 were returned, resulting in a 60% return rate. The A & D workers surveyed reflected the normal population in their belief in God and daily prayer, and similar to the clinical sample they had slightly more belief in a higher power. While the proportion of the professional sample who did not attend religious services reflected the normal population, there was nevertheless about twice the rate of attendance to worship by A & D workers. When the professional and clinical samples were compared on more specific variables, the evidence suggested that clinicians may in fact be more spiritually inclined, as a group, than their clients. There was a greater proportion of professionals who identified themselves as religious or spiritual and who meditated on a weekly basis or more regularly. Both groups had about 10% who read spiritual literature on a daily basis, but the clinical sample had significantly more who never read spiritual literature. The survey used was based upon a scale developed by Scott Tonigan and Bill Miller of the University of New Mexico, and had a range of possible scores from 0 to 40. Using the score as a measure of spirituality, I was able to look more closely at the professional sample. Out of a total 56 respondents, the mean was 24.7 and its standard deviation 10.2 Min 3, Max 41 ; . Two people declined to answer stating that the survey was biased in its use of "God". ; The comparison showed little difference between gender males slightly higher ; , or between Maori and Pakeha, but the few Pacific Islanders involved had noticeably higher scores. There was a trend that means of spiritual scores increased with age. But most interesting of all was the difference of means between the professions. Medicine, nursing and social work had markedly lower means than counselling and psychology; and the largest mean belonged to A & D clinicians who were working in the field because of their personal experience. The survey has limitations: one being that the scale used was more a measure of religiosity than spirituality, and another was the small numbers involved. But the results are informative. In general A & D workers do not seem any less religious than the normal population, nor less spiritually inclined than the clients they treat. In fact, with respect to meditation and attendance of worship they seem to be more active than the clinical population. However, some professions seem to be less religious than others, notably medicine and nursing. My thanks to everyone who took part in the survey. Michael Baker Galanter M; Research on Spirituality and Alcoholics Anonymous. Alcoholism: Clinical and Experimental Research. 1999, 23 4 ; : 716 - 719. Goldfarb L, Galanter M, McDowell D, Lifshutz H & Dermatis H; Medical Student and Patient attitudes Toward Religion an Spirituality in Recovery Process. American Journal of Drug and Alcohol Abuse. 1996, 22 4 ; : 549 - 561. Greeley A; Religion Not Dying Out Around the World the World. Origins. 1993, 23 4 ; : 49.
Several therapeutic modes are employed in the treatment of delirium and are discussed in this section. First, the cornerstone of treatment, psychiatric management, is defined and its components are described. Treatment of the delirium itself involves a set of environmental and supportive interventions and specific pharmacologic treatments. Environmental manipulations are generally designed to help reorient the patient and modulate the degree of stimulation. Supportive measures are designed to provide patient, family, and friends with both reassurance and education regarding the nature, temporal course, and sequelae of delirium and rythmol and pravastatin, for instance, pravastatin prescribing information.
Bupropion Hydrochloride Zyban ; , 150 mg Beclomethasone Dipropionate Vancenase ; AQ Nasal Fluticasone Propionate Flovent ; , 110 g MDI Ranitidine Hydrochloride, 150 mg Pravzstatin Sodium Pravachol ; , 20 mg Sertraline Hydrochloride Zoloft ; , 100 mg Nabumetone Relafen ; , 500 mg Paroxetine Hydrochloride Paxil ; , 20 mg Famotidine Pepcid ; , 20 mg Bupropion Hydrochloride Wellbutrin ; SR 150 Omeprazole Prilosec ; , 20 mg Loratadine Claritin ; , 10 mg Ciprofloxacin Hydrochloride Cipro ; , 500 mg Cefuroxime Axetil Ceftin ; , 250 mg Fluoxetine Hydrochloride Prozac ; , 20 mg Trazodone Hydrochloride, 100 mg Propoxyphene Hydrochloride and Acetaminophen Levothyroxine Levoxyl ; , 0.1 mg Atenolol, 50 mg Acetaminophen and Codeine Phosphate 300 30 Naproxen Sodium, 500 mg Amitriptyline Hydrochloride, 25 mg Glipizide Glucotrol ; XL, 10 mg Ibuprofen, 800 mg Estrogens, Conjugated and Medroxyprogesterone Acetate Prempro ; , 0.625 5 mg Levothyroxine Sodium Synthroid ; , 0.1 mg Hydrochlorothiazide, 25 mg Benazepril Hydrochloride Lotensin ; , 20 mg Estrogens, Conjugated Premarin ; , 0.625 mg Albuterol MDI 6 4 2.
ANTIRETROVIRALS NRTIs- abacavir Ziagen ; , abacavir lamivudine Epzicom ; , abacavir lamivudine zidovudine Trizivir ; , didanosine ddI, Videx EC ; , efavirenz emtricitabine tenofovir disproxil fumarate Atripla ; , emtricitabine Emtriva ; , lamivudine Epivir, 3TC ; , lamivudine zidovudine Combivir ; , stavudine d4T, Zerit ; , tenofovir Viread ; , tenofovir emtricitabine Truvada ; , zidovudine AZT, Retrovir ; . PIs- amprenavir Agenerase ; , atazanavir Reyataz ; , darunavir Prezista ; , fosamprenavir Lexiva ; , indinavir Crixivan ; , lopinavir ritonavir Kaletra ; , nelfinavir Viracept ; , ritonavir Norvir ; , saquinavir Fortovase, Invirase ; , tipranavir Aptivus ; . NNRTIs- delavirdine Rescriptor ; , efavirenz Sustiva ; , nevirapine Viramune ; . Entry Inhibitors- enfuvirtide Fuzeon ; . OI DRUGS PHS "A1 OI"s- acyclovir Zovirax ; , azithromycin Zithromax ; , clarithromycin Biaxin ; , fluconazole Diflucan ; , itraconazole Sporonox ; , leucovorin, pentamidine NebuPent, Pentam ; , pyramethamine Daraprim ; , rifabutin Mycobutin ; , sulfadiazine Microsulfon ; , TMP SMX Bactrim, Septra ; , valganciclovir Valcyte ; . Other OIsatovaquone Mepron ; , clotrimazole Mycelex, Gyne-Lotrimum ; , dapsone, ethambutol Myambutol ; , flucytosine Ancobon ; , ketoconazole Nizoral ; , metronidazole Flagyl ; , nystatin Mycostatin ; . ALL OTHERS atorvastatin Lipitor ; , fenofibrate Tricor ; , gemfibrozil Lopid ; , pravastatin Pravachol ; , oxandralone Oxandrin ; , testosterone, acetominophen hydrocodone Vicodin ; , amantadine Symmetrel ; , amitriptyline Elavil ; , bupropion Wellbutrin ; , buspirone BuSpar ; , carbamazepine Tegretol ; , cetaminophen + codeine Tylenol #3, Tylenol + codeine ; , chlorhexidine gluconate Peridex ; , clonidine hydrochloride ApoClonidine, Catapress, Nu-Clonidine ; , carbamazepine Tegretol ; , citalopram Celexa ; , desipramine Norpramine, Pertofrane ; , diphenhydramine Benadryl ; , diphenoxylate atropine Lomotil ; , esomeprazole magnesium Nexium ; , famotidine Pepcid ; , fluoxetine Prozac ; , gabapentin Neurontin ; , hydroxyzine Vistaril, Atarax ; , klonopin Clonazepam ; , lithium carbonate and pyrazinamide.
Impact on body composition. Thirty-three HIV-positive men on stable PI-containing HAART who had fasted cholesterol 6.5 mmol l were given dietary advice and started on a lipid-lowering diet for four weeks. They were then randomised to receive either pravastatin or placebo from week 4 to week 16. The primary endpoint of the study was time adjusted change in total cholesterol from baseline, with secondary endpoints looking at change in cholesterol from week 4, body composition using DEXA and abdominal CT, and additional lipid and cardiovascular risk markers. Results are summarised in Table 1 below. Table1: Pravastarin placebo p Chol AUC 0-16 week mmol l-1 wk ; 4-16 week mmol l-1 wk ; Total fat kg ; Limb fat kg ; % IAF - 0.6 - 0.82 + 1.03 + 0.72 - 2.90 -0.4 -0.34 -0.09 + 0.19 0.08.
Pravastatin patent litigation
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Long term intervention with pravastatin in ischaemic disease
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