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Send reprint requests to: Dr. Hitoshi Endou, Department of Pharmacology and Toxicology, Kyorin University School of Medicine, 6-20-2 Shinkawa, Mitaka, Tokyo 181-8611, Japan.
Anginal episodes, was detected in the right coronary artery RCA ; during coronary angiography. After sublingual nitroglycerin alleviated her symptoms and ST elevations, coronary angiography exhibited no organic stenotic lesions. She was diagnosed as having variant angina occurring at the RCA. Treated with oral nitrate and nifedipine, she remained generally well for 20 years. In June 1996, she suddenly died of acute myocardial infarction. Postmortem pathologic examination revealed severe stenotic lesions in the proximal segment of left anterior descending coronary artery with fresh thrombus. Diffuse minimal atherosclerotic lesions were observed in the RCA. Cross-sections of the RCA revealed diffuse intimal thickening with fibromuscular accumulation. With approval of the institutional review board on human research at Kobe University Hospital and informed consent from her family, pharmacologic and molecular experiments were performed using the coronary arteries of the patient. Proximal and distal portions of the RCA were used for recording isometric tension. Concentration-response curves for phenylephrine, 5-HT, sumatriptan, and ergonovine were determined by cumulative addition of each drug to the organ baths. In our previous study, 3 the threshold concentrations of isolated control human coronary arteries to 5-HT, ergonovine, and phenylephrine were reported to be as follows: 2.9 1.0 mol L; 8.7 3.1 10 mol L; and 2.9 0.9 10 mol L, respectively. These values are almost the same as those in another report.4 In the present study, 5-HT and ergonovine contracted helical strips of the RCA at extremely low concentrations: 10 12 mol L and 10 15 mol L, respectively. Interestingly, these coronary strips were also supersensitive to sumatriptan. The threshold concentration for sumatriptan was 10 12 mol L, which is much lower than that in a published study.4 These strips exhibited neither enhanced sensitivity nor reactivity to phenylephrine Fig 1 ; . However, the strip of left anterior descending coronary artery was supersensitive to none of these agents data not shown ; . We extracted total RNA from the coronary arteries of this patient after denuding the endothelium and performed RNase protection.
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Description technical field the present invention relates to a medical material such as a medical device which directly contacts with blood, and a process for producing the same.
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RISK FACTORS CONTRAINDICATIONS: 1. Do not use with ergotamine-containing products or within 2 weeks of an MAO-A inhibitor. 2. Do not use with patients with ischemic heart disease or uncontrolled blood pressure. 3. Do not use as a prophylactic agent. 4. Give only where diagnosis of migraine or cluster headache for injection ; is clearly established. 5. Patient should have a successful trial of sumartiptan in the medical office or emergency room to identify efficacy or possible side effects. 6. Contraindications to the use of 5-HT1 agonists: pregnancy, peripheral vascular disease i.e., thromboangitis, leutic arteritis, Raynaud's Syndrome, thrombophlebitis, arteriosclerosis ; , hepatic or renal impairment, coronary artery disease, uncontrolled hypertension. DRUG INTERACTIONS: Ergot-containing drugs have been reported to cause prolonged vasospastic reactions. Because there is a theoretical basis that these effects may be additive, use of ergotamine-containing or ergot-type medications like dihydroergotamine ; and 5-HT1 agonists within 24 hours of each other should be avoided. MAO-A inhibitors increase the systemic exposure of the 5-HT1 agonists and concomitant use is contraindicated. Concomitant use of more than one 5-HT1 agonist within 24 hours of each other is not recommended. Selective serotonin reuptake inhibitors SSRIs ; have been reported to cause weakness, hyperreflexia, and incoordination when coadministered with 5-HT1 agonists. MIGRAINE THERAPY OPTIONS: Table. Prophylactic therapy for migraine headache DRUG CLASS Beta Blockers.
These were frovatriptan frova ; vernalis ; and almotriptan axert ; pharmacia the trial results indicate that axert has similar efficacy to imitrex tablets sumatriptzn ; glaxosmithkline ; , a current leading anti-migraine medication and tetracycline.
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Sample Abstract - to show style COMPREHENSIVE THERAPEUTIC INTERCHANGE PROGRAM IN A TEACHING HOSPITAL Briscoe-Dwyer LA * , McCauley DL, Hoffman DM University Hospital Department of Pharmacy L3-559, Stony Brook, NY 11794-7310 This project describes a pharmacist-initiated dosage modification and therapeutic interchange program in a tertiary teaching hospital. A Medical Board-approved policy allows that medication orders may be modified and topamax.
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Bsessive-compulsive disorder OCD ; is a psychiatric condition with a lifetime prevalence of up to 3% The etiology of OCD is unknown, but data from challenge studies 2 ; and from pharmacological trials 3 ; have supported the involvement of the serotonergic system. Genetic studies have investigated possible variants of serotonergic system genes: the serotonin 5-HT ; receptor gene variants 5HT2A and 5-HT2C were found not to be associated to OCD 4, 5 ; , while mixed results were found for the serotonin transporter gene 6, 7 ; . The 5-HT1D receptor appears to be particularly interesting. It is a terminal autoreceptor, and challenge studies with nonselective ligands e.g., m-CPP ; 2 ; showed an acute worsening of obsessive-compulsive symptoms. In addition, a worsening of OCD symptoms has been reported following acute administration of sumatriptan, a selective agonist of the 5-HT1D receptor 2 ; . On the other hand, in OCD patients resistant to conventional pharmacotherapy, improvements have been reported with chronic administration of sumatriptan 8 ; . The aim of the current study was to investigate the presence of linkage disequilibrium between the 5-HT1D receptor gene 9 ; and OCD and tramadol.
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